SAB5200036
Monoclonal Anti-KCNT1 antibody produced in mouse
clone S3-26, 1 mg/mL, purified immunoglobulin
别名:
Anti-SLACK potassium channel, S3-26, Anti-potassium channel, subfamily T, member 1
产品名称
Monoclonal Anti-KCNT1 antibody produced in mouse, clone S3-26, 1 mg/mL, purified immunoglobulin
biological source
mouse
conjugate
unconjugated
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
S3-26, monoclonal
form
buffered aqueous glycerol solution
mol wt
antigen predicted mol wt 140 kDa
species reactivity
rat, human (weak), mouse
concentration
1 mg/mL
technique(s)
immunohistochemistry: suitable
western blot: suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Quality Level
Gene Information
rat ... Kcnt1(60444)
Biochem/physiol Actions
Detects ~140 kDa.
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Features and Benefits
Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.
Immunogen
Fusion protein amino acids 1168-1237 of rat Slo2.2 (Slack); NP_068625
Physical form
Solution in PBS, pH 7.4, 50% glycerol, and 0.09% sodium azide
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
常规特殊物品
此项目有
Duyen H Pham et al.
Human molecular genetics, 26(11), 2042-2052 (2017-03-24)
De novo and inherited mutations of X-chromosome cell adhesion molecule protocadherin 19 (PCDH19) cause frequent, highly variable epilepsy, autism, cognitive decline and behavioural problems syndrome. Intriguingly, hemizygous null males are not affected while heterozygous females are, contradicting established X-chromosome inheritance.
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