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Merck
CN

SML3731

Testosterone undecanoate

≥98% (HPLC)

别名:

(17β)-17-[(1-Oxoundecyl)oxy]androst-4-en-3-one, Undecanoic acid ester with testosterone

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关于此项目

经验公式(希尔记法):
C30H48O3
化学文摘社编号:
分子量:
456.70
UNSPSC Code:
51111800
NACRES:
NA.24
MDL number:
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产品名称

Testosterone undecanoate, ≥98% (HPLC)

assay

≥98% (HPLC)

form

powder

drug control

USDEA Schedule III

color

white to beige

solubility

DMSO: 2 mg/mL, clear (Warmed)

storage temp.

-10 to -25°C

Quality Level

Biochem/physiol Actions

Testosterone undecanoate is an orally available prodrug of testosterone that has a very long elimination half-life. It is an androgen and anabolic–androgenic steroid that acts as agonist of the androgen receptor (AR). Testosterone undecanoate is mainly used in the treatment of low testosterone levels in men. Also, it was approved for treatment of male hypogonadism.
orally available prodrug of testosterone that has a very long elimination half-life; androgen that acts as agonist of the androgen receptor (AR)

signalword

Danger

Hazard Classifications

Acute Tox. 4 Oral - Aquatic Acute 1 - Carc. 2 - Repr. 1B

存储类别

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Contribution of lymphatically transported testosterone undecanoate to the systemic exposure of testosterone after oral administration of two andriol formulations in conscious lymph duct-cannulated dogs
Journal of Pharmacology and Experimental Therapeutics, 306(3), 925-933 (2003)
Testosterone undecanoate and testosterone enanthate injections are both effective and safe in transmen over 5 years of administration
Clinical Endocrinology, 89(6), 878-886 (2018)
Atheer Zgair et al.
Molecules (Basel, Switzerland), 26(1) (2021-01-21)
Male hypogonadism is often treated by testosterone (T) replacement therapy such as oral administration of the ester prodrug, testosterone undecanoate (TU). However, the systemic exposure to T following oral TU is very low due to esterase-mediated metabolism, particularly in the

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