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Merck
CN

T0581

转谷氨酰胺酶 来源于天竺鼠

≥1.5 units/mg protein, recombinant, expressed in E. coli

别名:

TGase

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关于此项目

UNSPSC Code:
12352204
NACRES:
NA.54
Specific activity:
≥1.5 units/mg protein
Recombinant:
expressed in E. coli
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recombinant

expressed in E. coli

specific activity

≥1.5 units/mg protein

shipped in

dry ice

storage temp.

−20°C

Quality Level

Application

10 mM氯化钙用于激活酶。
转谷氨酰胺酶已用于一项可改善可量化分析研究,以充分表征转谷氨酰胺酶在亨廷顿氏病和阿尔茨海默病等疾病中的作用。转谷氨酰胺酶还用于研究转谷氨酰胺酶活性的非放射性斑点印迹试验。

Physical form

从10 mM NaH2PO4、150 mM NaCl、pH 8冻干。包含麦芽糖糊精。

Other Notes

一个单元,在pH6.0,37℃下,每分钟将催化Nα-Z-Gln-Gly和羟胺形成1.0 μ摩尔的异羟肟酸酯。(L-谷氨酸γ-单羟肟酸酯为参考标准。)

pictograms

Exclamation mark

signalword

Warning

hcodes

Hazard Classifications

Eye Irrit. 2

存储类别

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

常规特殊物品
此项目有

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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M Kelley et al.
Biochemical pharmacology, 35(2), 289-295 (1986-01-15)
The amino acid conjugation of the phenoxyherbicides 2,4-dichlorophenoxyacetate (2,4-D) and 2,4,5-trichlorophenoxyacetate (2,4,5-T) by animals was examined at the level of the enzymes catalyzing the reactions. The phenoxyherbicides were not substrates for the bile acid conjugating system but were substrates for
C M Becker et al.
Archives of biochemistry and biophysics, 223(2), 381-392 (1983-06-01)
Valproic acid (dipropylacetic acid), an antiepileptic agent known to be hepatotoxic in some patients, caused inhibition of lactate gluconeogenesis, fatty acid oxidation, and fatty acid synthesis by isolated hepatocytes. The latter process was the most sensitive to valproic acid, 50%
T S Emundianughe
Indian journal of experimental biology, 27(2), 160-162 (1989-02-01)
The metabolism of benzoic acid was examined in S. mansoni infected CBA mouse. The result showed that control animals dosed with 150 mg/kg benzoic acid resulted in urinary excretion of two metabolites, hippuric acid and benzoic acid glucuronide. Administration of
K D MacDermot et al.
Developmental pharmacology and therapeutics, 3(3), 150-159 (1981-01-01)
We report investigations of benzoate and glycine metabolism and glycine acyltransferase activity in rats. These studies provide insights related to the therapy and pathophysiology of human nonketotic hyperglycinemia. Liver acyltransferase activity increased sharply postnatally from low levels at birth, but
S Kølvraa et al.
Biochemical medicine and metabolic biology, 36(1), 98-105 (1986-08-01)
Prompted by the fact that the urinary excretion of organic acids in the riboflavin-deficient rat closely mimics that found in patients with inborn errors in the acyl-CoA dehydrogenation systems, the organelle localization and the apparent kinetic constants (Km and Vmax

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