产品名称
脂质标准品、单、双和甘油三酯混合物, certified reference material, pkg of 40 mg (Neat)
grade
certified reference material
TraceCERT®
product line
TraceCERT®
CofA
current certificate can be downloaded
packaging
pkg of 40 mg (Neat)
technique(s)
HPLC: suitable
gas chromatography (GC): suitable
application(s)
food and beverages
format
neat
functional group
ester
shipped in
dry ice
storage temp.
-10 to -25°C
Quality Level
Application
使用脂质标准品，单-、二- & 甘油三酯混合物可用于采用 TLC-FID 系统分析样品的脂质组成。它还用作采用二维 TLC 分析分级脂质的标准混合物。
有关合适的仪器技术的更多信息，请参阅产品的分析证书。如需进一步支持，请联系技术服务。
Other Notes
该认证参考物质（CRM）根据ISO 17034 和 ISO/IEC 17025生产和认证。有关此CRM使用的所有信息均可在分析证书上找到。
Legal Information
TraceCERT is a registered trademark of Merck KGaA, Darmstadt, Germany
存储类别
10 - Combustible liquids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
Triacylglycerol accumulation of Phaeodactylum tricornutum with different supply of inorganic carbon.
Peng, Xiaowei, et al.
Journal of Applied Phycology, 26.1, 131-139 (2014)
Efficient production of triacylglycerols rich in docosahexaenoic acid (DHA) by osmo-heterotrophic marine protists.
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Akhikun Nahar et al.
International journal of molecular sciences, 21(5) (2020-03-04)
In addition to cell membrane phospholipids, Actinobacteria in the order Corynebacteriales possess a waxy cell envelope containing mycolic acids (MA). In optimized culture condition, some species can also accumulate high concentrations of intracellular triacylglycerols (TAG), which are a potential source
Meei-Hua Lin et al.
Scientific reports, 9(1), 13254-13254 (2019-09-15)
Fatty acid transport protein 4 (FATP4) is an acyl-CoA synthetase that is required for normal permeability barrier in mammalian skin. FATP4 (SLC27A4) mutations cause ichthyosis prematurity syndrome, a nonlethal disorder. In contrast, Fatp4-/- mice die neonatally from a defective barrier.
Jin Young Huh et al.
Cell metabolism, 32(6), 1012-1027 (2020-11-06)
Hepatic TANK (TRAF family member associated NFκB activator)-binding kinase 1 (TBK1) activity is increased during obesity, and administration of a TBK1 inhibitor reduces fatty liver. Surprisingly, liver-specific TBK1 knockout in mice produces fatty liver by reducing fatty acid oxidation. TBK1
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