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Lithium chloride

Name: Lithium chloride
Brand: ALDRICH

powder, ≥99.98% trace metals basis

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Synonym(s):

Hydrochloric acid lithium salt

Linear Formula:

LiCl

CAS Number:

7447-41-8

Molecular Weight:

42.39

EC Number:

231-212-3

MDL number:

MFCD00011078

eCl@ss:

38050203

PubChem Substance ID:

24852187

NACRES:

NA.23

Quality Level

100

Assay

≥99.98% trace metals basis

form

powder

greener alternative product characteristics

Design for Energy Efficiency
Learn more about the Principles of Green Chemistry.

impurities

≤100.0 ppm Trace Metal Analysis

mp

605 °C (lit.)

solubility

H₂O: soluble

application(s)

battery manufacturing

greener alternative category

Enabling

SMILES string

[Li+].[Cl-]

InChI

1S/ClH.Li/h1H;/q;+1/p-1

InChI key

KWGKDLIKAYFUFQ-UHFFFAOYSA-M

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Related Categories

High-Purity Salts

General description

We are committed to bringing you Greener Alternative Products, which adhere to one or more of The 12 Principles of Greener Chemistry. This product has been enhanced for energy efficiency. Click here for more information.

Application

Lithium chloride can be used:

In the preparation of LiCl-PAM (polyacrylamide) ion-conducting neutral polymer electrolyte with ionic conductivity >10 mS cm⁻¹ .
As an efficient catalyst in the cyanosilylation of aldehydes and ketones to the corresponding silylated cyanohydrins.
As a reaction medium in combination with N,N-dimethylacetamide for the esterification of cellulose.

Pictograms

GHS07

Signal Word

Warning

Hazard Statements

H302 - H315 - H319

Precautionary Statements

P264 - P280 - P301 + P312 - P302 + P352 - P305 + P351 + P338 - P332 + P313

Hazard Classifications

Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2

Storage Class Code

13 - Non Combustible Solids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Lithium chloride corrects weakness and myopathology in a preclinical model of LGMD1D.

Andrew R Findlay et al.

Neurology. Genetics, 5(2), e318-e318 (2019-05-28)

To understand DNAJB6's function in skeletal muscle and identify therapeutic targets for limb-girdle muscular dystrophy 1D (LGMD1D). DNAJB6 knockout (KO) myoblasts were generated with Crispr/cas9 technology, and differentially accumulated proteins were identified using stable isotope labeling, followed by quantitative mass

Wnt affects symmetry and morphogenesis during post-embryonic development in colonial chordates.

Alessandro Di Maio et al.

EvoDevo, 6, 17-17 (2015-07-15)

Wnt signaling is one of the earliest and most highly conserved regulatory pathways for the establishment of the body axes during regeneration and early development. In regeneration, body axes determination occurs independently of tissue rearrangement and early developmental cues. Modulation

Phosphorylation of amyloid precursor protein at threonine 668 is essential for its copper-responsive trafficking in SH-SY5Y neuroblastoma cells.

Karla M Acevedo et al.

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Amyloid precursor protein (APP) undergoes post-translational modification, including O- and N-glycosylation, ubiquitination, and phosphorylation as it traffics through the secretory pathway. We have previously reported that copper promotes a change in the cellular localization of APP. We now report that

Application of scaffolds for bone regeneration strategies: current trends and future directions.

Gry Hulsart Billström et al.

Injury, 44 Suppl 1, S28-S33 (2013-02-13)

Scaffolds are extensively used in surgery to replace missing bone and to achieve bony union and fusion. An ideal scaffold should not only maintain, induce, and restore biological functions where cells, extracellular matrix, and growth factors are needed, but also

BDNF signaling in the VTA links the drug-dependent state to drug withdrawal aversions.

Hector Vargas-Perez et al.

The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(23), 7899-7909 (2014-06-06)

Drug administration to avoid unpleasant drug withdrawal symptoms has been hypothesized to be a crucial factor that leads to compulsive drug-taking behavior. However, the neural relationship between the aversive motivational state produced by drug withdrawal and the development of the

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