KEY465194751
Key Organics / BIONET
ethyl N-(3-oxobutanoyl)carbamate
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3 X 1.2 ML
CN¥904.05
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3 X 1.2 ML
CN¥904.05
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CN¥904.05
Please contact Customer Service for Availability
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Iupac Name
ethyl acetoacetylcarbamate
Inchi Code
1S/C7H11NO4/c1-3-12-7(11)8-6(10)4-5(2)9/h3-4H2,1-2H3,(H,8,10,11)
InChI key
KTKYFDJHFHNAOV-UHFFFAOYSA-N
Purity
90%
Country of Origin
GB
Physical Form
Solid
Product Link
https://www.keyorganics.net/sku/3E-900
Signal Word
Warning
Hazard Codes
MSDS
https://www.keyorganics.net/sds/3E-900
Regulatory Information
易制毒化学品（3类）
危险化学品
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Issues in Pharmacology, Pharmacy, Drug Research, and Drug Innovation: 2011 Edition (2012)
Yang Cao et al.
British journal of haematology, 168(5), 701-707 (2014-11-06)
CXCR4(WHIM) frameshift and nonsense mutations follow MYD88(L265P) as the most common somatic variants in Waldenström Macroglobulinaemia (WM), and impact clinical presentation and ibrutinib response. While the nonsense (CXCR4(S338X) ) mutation has been investigated, little is known about CXCR4 frameshift (CXCR4(FS)
Federico Mele et al.
Nature communications, 6, 6431-6431 (2015-03-17)
T helper (TH) cell polarization during priming is modulated by a number of signals, but whether polarization to a given phenotype also influences recall responses of memory TH cells is relatively unknown. Here we show that miR-181a is selectively induced
Inna V Fedorenko et al.
Proteomics, 15(2-3), 327-339 (2014-10-24)
Basal and kinase inhibitor driven adaptive signaling has been examined in a panel of melanoma cell lines using phosphoproteomics in conjunction with pathway analysis. A considerable divergence in the spectrum of tyrosine-phosphorylated peptides was noted at the cell line level.
L Schafranek et al.
Leukemia, 29(1), 76-85 (2014-05-13)
Kinase inhibitors block proliferative signals in BCR-ABL1+ leukemic cells, but their capacity to induce apoptosis is poorly understood. Initial studies suggested that very brief exposure to kinase inhibitors was sufficient to induce apoptosis in chronic myeloid leukemia (CML) cells. However
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