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PRS3771

Sigma-Aldrich

Anti-HRK antibody produced in rabbit

affinity isolated antibody, buffered aqueous solution

Synonym(s):
Anti-Harakiri, BCL2 Interacting protein (contains only BH3 domain), Anti-Harakiri, Anti-Neuronal death proteinDP
NACRES:
NA.41

biological source

rabbit

Quality Level

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

predicted mol wt 10 kDa

species reactivity

mouse, human

technique(s)

indirect ELISA: suitable
western blot: suitable

UniProt accession no.

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... HRK(8739)

Immunogen

a 15 amino acid peptide from near the center of human Hrk.

Linkage

The action of this antibody can be blocked using blocking peptide SBP3771.

Physical form

Solution in phosphate buffered saline containing 0.02% sodium azide

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificate of Analysis

Certificate of Origin

Kinnosuke Yahiro et al.
Cell death discovery, 7(1), 360-360 (2021-11-21)
Locus for Enterocyte Effacement (LEE)-positive Shiga-toxigenic Escherichia coli (STEC) contributes to many global foodborne diseases, with infection characterized by severe gastrointestinal symptoms, including bloody diarrhea. The incidence of LEE-negative STEC-mediated disease is also increasing globally. Subtilase cytotoxin (SubAB) is released
Jessica F Boehler et al.
The Journal of pathology, 249(2), 215-226 (2019-05-29)
The etiology of myositis is unknown. Although attempts to identify viruses in myositis skeletal muscle have failed, several studies have identified the presence of a viral signature in myositis patients. Here we postulate that in individuals with susceptible genetic backgrounds
Erica Anspach Will et al.
Biology of reproduction, 96(4), 843-854 (2017-04-04)
The present studies were designed to determine whether progesterone (P4)-progesterone receptor membrane component 1 (PGRMC1) signaling is able to attenuate the apoptotic effects of oxidative stress induced by hydrogen peroxide (H2O2). To achieve this goal, freshly isolated human granulosa/luteal cells

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