SML3731
Testosterone undecanoate
≥98% (HPLC)
Synonym(s):
(17β)-17-[(1-Oxoundecyl)oxy]androst-4-en-3-one, Undecanoic acid ester with testosterone
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About This Item
Empirical Formula (Hill Notation):
C30H48O3
CAS Number:
Molecular Weight:
456.70
UNSPSC Code:
51111800
NACRES:
NA.24
MDL number:
Product Name
Testosterone undecanoate, ≥98% (HPLC)
assay
≥98% (HPLC)
form
powder
drug control
USDEA Schedule III
color
white to beige
solubility
DMSO: 2 mg/mL, clear (Warmed)
storage temp.
-10 to -25°C
Quality Level
Related Categories
Biochem/physiol Actions
Testosterone undecanoate is an orally available prodrug of testosterone that has a very long elimination half-life. It is an androgen and anabolic–androgenic steroid that acts as agonist of the androgen receptor (AR). Testosterone undecanoate is mainly used in the treatment of low testosterone levels in men. Also, it was approved for treatment of male hypogonadism.
orally available prodrug of testosterone that has a very long elimination half-life; androgen that acts as agonist of the androgen receptor (AR)
signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 4 Oral - Aquatic Acute 1 - Carc. 2 - Repr. 1B
Storage Class
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
Regulatory Information
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Contribution of lymphatically transported testosterone undecanoate to the systemic exposure of testosterone after oral administration of two andriol formulations in conscious lymph duct-cannulated dogs
Journal of Pharmacology and Experimental Therapeutics, 306(3), 925-933 (2003)
Testosterone undecanoate and testosterone enanthate injections are both effective and safe in transmen over 5 years of administration
Clinical Endocrinology, 89(6), 878-886 (2018)
Atheer Zgair et al.
Molecules (Basel, Switzerland), 26(1) (2021-01-21)
Male hypogonadism is often treated by testosterone (T) replacement therapy such as oral administration of the ester prodrug, testosterone undecanoate (TU). However, the systemic exposure to T following oral TU is very low due to esterase-mediated metabolism, particularly in the
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