201-469-6

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Biophysical defects of an SCN5A V1667I mutation associated with epinephrine-induced marked QT prolongation.

Tadashi Nakajima et al.

Journal of cardiovascular electrophysiology, 31(8), 2107-2115 (2020-05-22)

The epinephrine infusion test (EIT) typically induces marked QT prolongation in LQT1, but not LQT3, while the efficacy of β-blocker therapy is established in LQT1, but not LQT3. We encountered an LQT3 family, with an SCN5A V1667I mutation, that exhibited

Inhibited neurogenesis in JNK1-deficient embryonic stem cells.

Claudia R Amura et al.

Molecular and cellular biology, 25(24), 10791-10802 (2005-11-30)

The JNKs are components of stress signaling pathways but also regulate morphogenesis and differentiation. Previously, we invoked a role for the JNKs in nerve growth factor (NGF)-stimulated PC12 cell neural differentiation (L. Marek et al., J. Cell. Physiol. 201:459-469, 2004;

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