286-94-2

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Showing 1-5 of 5 results for "286-94-2" within Papers

An association study on polymorphisms in the PEA15, ENTPD4, and GAS2L1 genes and schizophrenia.

Atsushi Saito et al.

Psychiatry research, 185(1-2), 9-15 (2010-06-12)

Our previous study examined a number of methamphetamine (METH)/phencyclidine (PCP)-reactive tags in rat brain, using a serial analysis of gene expression. Among human homologous genes, which matched METH/PCP-reactive tags, three human genes were identified: phosphoprotein enriched in astrocyte 15 (PEA15)

Molecular cloning of magnesium-independent type 2 phosphatidic acid phosphatases from airway smooth muscle.

R J Tate et al.

Cellular signalling, 11(7), 515-522 (1999-07-16)

Members of the type 2 phosphatidic acid phosphatase (PAP2) family catalyse the dephosphorylation of phosphatidic acid (PA), lysophosphatidate and sphingosine 1-phosphate. Here, we demonstrate the presence of a Mg(2+)-independent and N-ethymaleimide-insensitive PAP2 activity in cultured guinea-pig airway smooth muscle (ASM)

Common alleles in candidate susceptibility genes associated with risk and development of epithelial ovarian cancer.

Maria Notaridou et al.

International journal of cancer, 128(9), 2063-2074 (2010-07-17)

Common germline genetic variation in the population is associated with susceptibility to epithelial ovarian cancer. Microcell-mediated chromosome transfer and expression microarray analysis identified nine genes associated with functional suppression of tumorogenicity in ovarian cancer cell lines; AIFM2, AKTIP, AXIN2, CASP5

Influences of thiopurine methyltransferase genotype and activity on thiopurine-induced leukopenia in Korean patients with inflammatory bowel disease: a retrospective cohort study.

Jae Hak Kim et al.

Journal of clinical gastroenterology, 44(10), e242-e248 (2010-03-24)

Myelotoxicity has been shown to be very common in Korean patients with inflammatory bowel disease (IBD) during azathioprine (AZA) or 6-mercaptopurine (6-MP) treatment. The purpose of this study was to investigate the relative risk of the thiopurine methyltransferase (TPMT) and

Molecular cloning and functional expression of human 3-hydroxyanthranilic-acid dioxygenase.

P Malherbe et al.

The Journal of biological chemistry, 269(19), 13792-13797 (1994-05-13)

Increased cerebral levels of the endogenous excitotoxin quinolinic acid (QUIN) have been speculatively linked to neuronal damage following neurological and inflammatory disorders. 3-Hydroxyanthranilic-acid dioxygenase (3-HAO; 3-hydroxyanthranilate 3,4-dioxygenase, EC 1.13.11.6) is the enzyme that catalyzes the synthesis of QUIN from 3-hydroxyanthranilic

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