Yuanyuan Zhang et al.
Advanced science (Weinheim, Baden-Wurttemberg, Germany), 10(7), e2206958-e2206958 (2023-01-03)
Development of abdominal aortic aneurysms (AAA) enhances lesion group-2 innate lymphoid cell (ILC2) accumulation and blood IL5. ILC2 deficiency in Rorafl/fl Il7rCre/+ mice or induced ILC2 depletion in Icosfl-DTR-fl/+ Cd4Cre/+ mice expedites AAA growth, increases lesion inflammation, but leads to