05-782
抗GST标签抗体
Upstate®, from mouse
别名:
Anti-DFN7, Anti-FAEES3, Anti-GST3, Anti-GSTP, Anti-HEL-S-22, Anti-PI
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关于此项目
UNSPSC Code:
12352203
NACRES:
NA.43
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
monoclonal
Application:
WB
Citations:
19
biological source
mouse
conjugate
unconjugated
antibody form
purified antibody
antibody product type
primary antibodies
clone
monoclonal
species reactivity
bacteria
manufacturer/tradename
Upstate®
technique(s)
western blot: suitable
isotype
IgG
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Quality Level
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General description
因识别的GST融合蛋白而异。
Immunogen
来自日本血吸虫、在大肠杆菌中表达的重组 GST （谷胱甘肽S转移酶）
Application
使用经验证可用于 WB 的抗-GST 标签抗体检测GST 标签。
Biochem/physiol Actions
GST 融合蛋白
预计不与其它物种的GST交叉反应。
Physical form
形式：纯化
蛋白G纯化的小鼠IgG，溶于储存缓冲液（含0.05% 叠氮化钠和30%甘油的PBS）。
Analysis Note
通过免疫印迹法对重组GST蛋白（货号12-350）和GST-Abltide融合蛋白（货号12-525）进行了常规评估
Legal Information
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
12 - Non Combustible Liquids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
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The Journal of physiology, 590(10), 2333-2351 (2012-03-28)
Rapid exchange of metabolites between different cell types is crucial for energy homeostasis of the brain. Besides glucose, lactate is a major metabolite in the brain and is primarily produced in astrocytes. In the present study, we report that carbonic
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Michael Vershinin, Jing Xu, David S Razafsky, Stephen J King, Steven P Gross
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Stuart Sullivan et al.
Nature communications, 12(1), 6129-6129 (2021-10-23)
Polarity underlies all directional growth responses in plants including growth towards the light (phototropism). The plasma-membrane associated protein, NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3) is a key determinant of phototropic growth which is regulated by phototropin (phot) AGC kinases. Here we demonstrate
Katrina Montales et al.
PloS one, 17(8), e0271905-e0271905 (2022-08-03)
Ataxia Telangiectasia mutated and RAD3-related (ATR) kinase is activated by DNA replication stress and also by various forms of DNA damage, including DNA double-strand breaks (DSBs). Recruitment to sites of damage is insufficient for ATR activation as one of two
Katrina Montales et al.
Scientific reports, 11(1), 467-467 (2021-01-13)
The protein kinase ATR is activated at sites of DNA double-strand breaks where it plays important roles in promoting DNA end resection and regulating cell cycle progression. TOPBP1 is a multi BRCT repeat containing protein that activates ATR at DSBs.
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