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Merck
CN

AB3542

抗单羧酸转运蛋白2抗体

serum, Chemicon®

别名:

MCT2

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关于此项目

UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
polyclonal
Application:
ELISA, WB
Citations:
11
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biological source

rabbit

Quality Level

conjugate

unconjugated

antibody form

serum

antibody product type

primary antibodies

clone

polyclonal

species reactivity

rat

manufacturer/tradename

Chemicon®

technique(s)

ELISA: suitable, western blot: suitable

NCBI accession no.

UniProt accession no.

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... SLC16A7(9194)

Immunogen

大鼠MCT2 C末端附近的15个氨基酸肽序列(ackson et al. 1997; Dao et al. 1998; Koehler-Stec et al. 1998; Price et al. 1998; Halestrap & Price 1999)。

Application

免疫印迹分析

ELISA:1:10,000-1:100,000,每孔使用50-100 ng对照肽(货号AG218)。

最佳工作稀释度必须由最终用户进行确定。
抗单羧酸转运蛋白2抗体是针对单羧酸转运蛋白2的抗体,用于ELISA& WB。
研究子类别
离子通道 & 转运蛋白
研究类别
神经科学

Biochem/physiol Actions

可识别大鼠单羧酸转运蛋白2(MCT2)。免疫原与其他MCT没有明显的序列同源性。

免疫原序列在小鼠中93%保守,在仓鼠中66%保守。与其他物种的反应性尚未得到确认。

Physical form

兔血清。 液体。

Preparation Note

以未稀释的等分试样保存于-20°C下6个月。应避免反复冻/融循环。

Other Notes

替代:AB3542P

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

除非我们的产品目录或产品附带的其他公司文档另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。


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存储类别

10 - Combustible liquids

wgk

WGK 1



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Estrogen Receptor Involvement in Noradrenergic Regulation of Ventromedial Hypothalamic Nucleus Glucoregulatory Neurotransmitter and Stimulus-Specific Glycogen Phosphorylase Enzyme Isoform Expression.
A S M H Mahmood et al.
ASN neuro, 12, 1759091420910933-1759091420910933 (2020-04-03)
J S Gilbert et al.
Placenta, 33(2), 100-105 (2011-12-22)
While utero-placental insufficiency is associated with adverse outcomes for both mother and fetus, many of the maternal-fetal adaptations during pregnancy in models of fetal compromise remain unclear. The purpose of this study was to determine if chronically reduced uterine perfusion
Emmanuel Cruz et al.
Glia, 70(11), 2207-2231 (2022-08-03)
The consumption of glucose in the brain peaks during late childhood; yet, whether and how glucose metabolism is differentially regulated in the brain during childhood compared to adulthood remains to be understood. In particular, it remains to be determined how



全球贸易项目编号

货号GTIN
AB354204053252675300