产品名称
抗-聚集蛋白抗体, Chemicon®, from mouse
biological source
mouse
conjugate
unconjugated
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
monoclonal
species reactivity
mouse, rat
manufacturer/tradename
Chemicon®
technique(s)
immunocytochemistry: suitable
western blot: suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Quality Level
Gene Information
human ... AGRN(375790)
Application
免疫细胞化学：10 μg/mL
最终工作稀释度必须由最终用户确定。
最终工作稀释度必须由最终用户确定。
抗凝集素抗体是用于WB & IC的抗凝集素抗体。
研究子类别
生长锥&轴突导向
突触&突触生物学
生长锥&轴突导向
突触&突触生物学
研究类别
神经科学
神经科学
Biochem/physiol Actions
MAB5204识别大鼠和小鼠凝集素。该表位已定位在剪接位点Z附近。该抗体与凝集素的结合导致nAChR簇显著减少。
Disclaimer
除非我们的目录或产品随附的其他公司文件中另有说明，否则我们的产品预期仅用于研究用途，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或对人类或动物的任何类型的消费或应用。
Immunogen
重组大鼠凝集素（C末端构建体）
Other Notes
浓度：关于批次特定浓度请参见检验报告。
Physical form
形式：纯化
纯化的免疫球蛋白。 液体形式，溶于含50%甘油和0.09%叠氮化钠的PBS中。
Preparation Note
在-20°C或更低温度下保持冻干状态长达12个月。复溶后，以未稀释的等分试样在-20°C至-70°C下保存长达6个月。应避免反复冻/融循环。
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
未找到合适的产品？
试试我们的产品选型工具.
存储类别
10 - Combustible liquids
wgk
WGK 2
Nandor Nagy et al.
Development (Cambridge, England), 145(9) (2018-04-22)
The enteric nervous system (ENS) arises from neural crest cells that migrate, proliferate, and differentiate into enteric neurons and glia within the intestinal wall. Many extracellular matrix (ECM) components are present in the embryonic gut, but their role in regulating
Jessica L Mueller et al.
Stem cells translational medicine, 13(5), 490-504 (2024-02-22)
Regenerative cell therapy to replenish the missing neurons and glia in the aganglionic segment of Hirschsprung disease represents a promising treatment option. However, the success of cell therapies for this condition are hindered by poor migration of the transplanted cells.
The COOH-terminal domain of agrin signals via a synaptic receptor in central nervous system neurons.
Hoover, CL; Hilgenberg, LG; Smith, MA
The Journal of cell biology null
Paul J Thomas et al.
The American journal of pathology, 186(9), 2429-2448 (2016-08-27)
Overexpression of B4GALNT2 (previously GALGT2) inhibits the development of muscle pathology in mouse models of Duchenne muscular dystrophy, congenital muscular dystrophy 1A, and limb girdle muscular dystrophy 2D. In these models, muscle GALGT2 overexpression induces the glycosylation of α dystroglycan
Zhenxi Zhang et al.
Proceedings of the National Academy of Sciences of the United States of America, 110(48), 19348-19353 (2013-11-06)
The motor neuron (MN) degenerative disease, spinal muscular atrophy (SMA) is caused by deficiency of SMN (survival motor neuron), a ubiquitous and indispensable protein essential for biogenesis of snRNPs, key components of pre-mRNA processing. However, SMA's hallmark MN pathology, including
我们的科学家团队拥有各种研究领域经验，包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.
联系客户支持