产品名称
抗 兔 IgG(全分子)–FITC 山羊抗, affinity isolated antibody, buffered aqueous solution
biological source
goat
conjugate
FITC conjugate
antibody form
affinity isolated antibody
antibody product type
secondary antibodies
clone
polyclonal
form
buffered aqueous solution
technique(s)
indirect immunofluorescence: 1:80
storage temp.
2-8°C
target post-translational modification
unmodified
Quality Level
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Application
抗兔 IgG(全分子)-FITC 抗体适用于免疫荧光 。
Biochem/physiol Actions
结合所有兔子免疫球蛋白 G。
Disclaimer
除非我们的产品目录或产品随附的其他公司文件中另有说明,否则我们的产品仅用于研究用途,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、体外或体内治疗用途或任何类型的人类或动物消费或应用。
General description
IgG 是糖蛋白抗体,可调节几种免疫反应。针对靶蛋白的兔 IgG 常被用作各种研究应用的一抗。因此,与可检测底物偶联的二级抗兔 IgG 是分析靶蛋白的有用工具。抗兔 IgG(全分子)-FITC 抗体与兔血清和兔 IgG 反应。
Immunogen
纯化兔 IgG
Physical form
0.01 M 磷酸盐缓冲液 (pH 7.4) 中的溶液,含 1% 牛血清白蛋白和 15 mM 叠氮化钠
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存储类别
10 - Combustible liquids
wgk
nwg
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
常规特殊物品
此项目有
M J Laskowski
Planta, 181(1), 44-52 (1990-04-01)
The orientation of microtubules in cells of redlight-grown pea plants (Pisum sativum L.) was examined by means of immunofluorescence. Microtubules (MTs) in rapidly elongating, subepidermal cells commonly form multiple, parallel strands that run transverse to the cell's axis of elongation.
Claire Joly Condette et al.
PloS one, 9(7), e102217-e102217 (2014-07-16)
The epithelium's barrier function is crucial for maintaining homeostasis and preventing the passage of food antigens and luminal bacteria. This function is essentially subserved by tight junctions (TJs), multiprotein complexes located in the most apical part of the lateral membrane.
L K Zhuang et al.
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MicroRNA (miRNA) and long non-coding RNA (lncRNA) have been demonstrated to participate in the progression of many cancers. Hepatocellular carcinoma (HCC) is one of the most common and aggressive malignant tumors worldwide, while the molecular mechanisms underlying HCC tumorigenesis are
M A Sussman et al.
The Journal of clinical investigation, 101(1), 51-61 (1998-02-14)
Loss of myofibril organization is a common feature of chronic dilated and progressive cardiomyopathy. To study how the heart compensates for myofibril degeneration, transgenic mice were created that undergo progressive loss of myofibrils after birth. Myofibril degeneration was induced by
Ga Bin Park et al.
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