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Merck
CN

SAB2702217

单克隆抗 HA 标签 小鼠抗

clone GT4810, affinity isolated antibody

别名:

Mouse Anti-hemagglutinin

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关于此项目

NACRES:
NA.43
UNSPSC Code:
12352203
Conjugate:
unconjugated
Clone:
GT4810, monoclonal
Application:
IF, IP, WB
Citations:
3
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biological source

mouse

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

GT4810, monoclonal

form

buffered aqueous solution

concentration

1mg/mL

technique(s)

immunoprecipitation (IP): suitable, indirect immunofluorescence: suitable, western blot: 1000-10000

isotype

IgG2a

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Quality Level

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Immunogen

用于生成该对应于HA标签抗体的免疫原

Application

建议的起始稀释度如下:ICC/IF:1:100-1:1000,IP: 1:100-1:500,蛋白质印迹:1:1000-1:10000.尚未在其他应用中测试。最佳工作稀释度应由最终用户通过实验确定。

Features and Benefits

完放心地使用我们的抗体。如果抗体在您的申请的研究中不起作用,我们将全额退款或安排替代抗体。了解更多信息。

Physical form

磷酸盐缓冲液,未加保护剂。

Other Notes

纯化方法:Protein G亲和纯化

Disclaimer

除非我们的产品目录或产品附带的其他公司文档另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。

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存储类别

12 - Non Combustible Liquids

wgk

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

常规特殊物品
低风险生物材料
此项目有

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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访问文档库

Janik Puttemans et al.
Molecular cancer therapeutics (2021-10-21)
To this day, multiple myeloma remains an incurable cancer. For many patients, recurrence is unavoidably a result of lacking treatment options in the minimal residual disease stage. This is due to residual and treatment-resistant myeloma cells that can cause disease
Ben Zhou et al.
Cell, 177(2), 299-314 (2019-04-02)
Autophagy is required in diverse paradigms of lifespan extension, leading to the prevailing notion that autophagy is beneficial for longevity. However, why autophagy is harmful in certain contexts remains unexplained. Here, we show that mitochondrial permeability defines the impact of
Antonio Galeone et al.
eLife, 9 (2020-07-29)
During endoplasmic reticulum-associated degradation (ERAD), the cytoplasmic enzyme N-glycanase 1 (NGLY1) is proposed to remove N-glycans from misfolded N-glycoproteins after their retrotranslocation from the ER to the cytosol. We previously reported that NGLY1 regulates Drosophila BMP signaling in a tissue-specific

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