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Merck
CN

13401

Sodium

≥99%, pieces (stored under mineral oil, heavy)

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About This Item

Empirical Formula (Hill Notation):
Na
CAS Number:
Molecular Weight:
22.99
UNSPSC Code:
12352000
PubChem Substance ID:
MDL number:
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InChI

1S/Na

SMILES string

[Na]

InChI key

KEAYESYHFKHZAL-UHFFFAOYSA-N

assay

≥99%

form

pieces (stored under mineral oil, heavy)

reaction suitability

reagent type: reductant

resistivity

4.69 μΩ-cm, 20°C

bp

883 °C (lit.)

mp

97.8 °C (lit.)

anion traces

chloride (Cl-): ≤100 mg/kg

cation traces

Ca: ≤1000 mg/kg, K: ≤500 mg/kg

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pictograms

FlameCorrosion

signalword

Danger

hcodes

supp_hazards

Storage Class

4.3 - Hazardous materials which set free flammable gases upon contact with water

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Hazard Classifications

Skin Corr. 1B - Water-react. 1

Regulatory Information

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Andrew Mente et al.
The New England journal of medicine, 371(7), 601-611 (2014-08-15)
Higher levels of sodium intake are reported to be associated with higher blood pressure. Whether this relationship varies according to levels of sodium or potassium intake and in different populations is unknown. We studied 102,216 adults from 18 countries. Estimates
Supriya S Jalimarada et al.
Investigative ophthalmology & visual science, 54(6), 4330-4340 (2013-06-08)
Mutations in SLC4A11, a member of the SLC4 superfamily of bicarbonate transporters, give rise to corneal endothelial cell dystrophies. SLC4A11 is a putative Na⁺ borate and Na⁺:OH⁻ transporter. Therefore we ask whether SLC4A11 in corneal endothelium transports borate (B[OH]₄⁻), bicarbonate
Nicole Li et al.
American heart journal, 166(5), 815-822 (2013-11-02)
Cardiovascular diseases are the leading cause of death and disability in China. High blood pressure caused by excess intake of dietary sodium is widespread and an effective sodium reduction program has potential to improve cardiovascular health. This study is a
Matthew R Whorton et al.
Nature, 498(7453), 190-197 (2013-06-07)
G-protein-gated inward rectifier K(+) (GIRK) channels allow neurotransmitters, through G-protein-coupled receptor stimulation, to control cellular electrical excitability. In cardiac and neuronal cells this control regulates heart rate and neural circuit activity, respectively. Here we present the 3.5 Å resolution crystal structure
Etana Padan
Biochimica et biophysica acta, 1837(7), 1047-1062 (2013-12-24)
The crystal structure of down-regulated NhaA crystallized at acidic pH4 [21] has provided the first structural insights into the antiport mechanism and pH regulation of a Na(+)/H(+) antiporter [22]. On the basis of the NhaA crystal structure [21] and experimental

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