222909
N-Phenylthiourea
97%
Synonym(s):
1-Phenyl-2-thiourea, PTU, Phenylthiocarbamide
Sign In to View Organizational & Contract Pricing.
Select a Size
Change View
About This Item
Linear Formula:
C6H5NHCSNH2
CAS Number:
Molecular Weight:
152.22
EC Number:
203-151-2
UNSPSC Code:
12352100
PubChem Substance ID:
Beilstein/REAXYS Number:
907309
MDL number:
assay
97%
mp
145-150 °C (lit.)
SMILES string
NC(=S)Nc1ccccc1
InChI
1S/C7H8N2S/c8-7(10)9-6-4-2-1-3-5-6/h1-5H,(H3,8,9,10)
InChI key
FULZLIGZKMKICU-UHFFFAOYSA-N
Still not finding the right product?
Explore all of our products under N-Phenylthiourea
signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 1 Oral - Skin Sens. 1
Storage Class
6.1B - Non-combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges
Regulatory Information
新产品
This item has
Choose from one of the most recent versions:
Already Own This Product?
Find documentation for the products that you have recently purchased in the Document Library.
Anna A Brozyna et al.
International journal of cancer, 123(6), 1448-1456 (2008-06-21)
Melanin pigment displays strong photo- and radioprotective properties, suggesting that inhibition melanogenesis could increase sensitivity of melanoma to ionizing radiation. We tested this concept in human melanoma cells cultured in either Ham's F10 medium to maintain amelanotic phenotype or DMEM
Xiangguo Shi et al.
Blood, 125(19), 2974-2984 (2015-03-18)
Isocitrate dehydrogenase 1 mutation (IDH1-R132H) was recently identified in acute myeloid leukemia with normal cytogenetics. The mutant enzyme is thought to convert α-ketoglutarate to the pathogenic 2-hydroxyglutarate (2-HG) that affects DNA methylation via inhibition of ten-eleven translocation 2. However, the
Marco Venturin et al.
Journal of medical genetics, 51(7), 436-443 (2014-04-09)
Cardiovascular malformations have a higher incidence in patients with NF1 microdeletion syndrome compared to NF1 patients with intragenic mutation, presumably owing to haploinsufficiency of one or more genes included in the deletion interval and involved in heart development. In order