298670
DL-Cystine
99%
Synonym(s):
(±)-3,3′-Dithiobis(2-aminopropionicacid)
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About This Item
Linear Formula:
HOOCCH(NH2)CH2SSCH2CH(NH2)COOH
CAS Number:
Molecular Weight:
240.30
EC Number:
213-094-5
UNSPSC Code:
12352103
PubChem Substance ID:
Beilstein/REAXYS Number:
1728095
MDL number:
InChI key
LEVWYRKDKASIDU-UHFFFAOYSA-N
InChI
1S/C6H12N2O4S2/c7-3(5(9)10)1-13-14-2-4(8)6(11)12/h3-4H,1-2,7-8H2,(H,9,10)(H,11,12)
SMILES string
NC(CSSCC(N)C(O)=O)C(O)=O
assay
99%
mp
227 °C (dec.) (lit.)
Storage Class
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
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Song Li et al.
Chemistry, an Asian journal, 14(12), 2155-2161 (2019-04-27)
Reactions coupled self-assembly represents a step forward towards biomimetic behavior in the field of supramolecular research. Here, two pH-dependent reactions of thiol-disulfide exchange and ligand exchange were used to couple with the self-assembly of an AuI -thiolate coordination polymer consisting
Maria José Alves et al.
Journal of medical microbiology, 63(Pt 3), 471-477 (2014-01-17)
A group of biofilm-producing bacteria isolated from patients with urinary tract infections was evaluated, identifying the main factors contributing to biofilm formation. Among the 156 isolates, 58 (37.2%) were biofilm producers. The bacterial species (P<0.001), together with patient's gender (P
E Patricia Owen et al.
Pediatric nephrology (Berlin, Germany), 30(4), 595-601 (2014-10-19)
The mutations responsible for cystinosis in South African patients are currently unknown. A pertinent question is whether they are similar to those described elsewhere in the world. Children who were being managed for cystinosis in the Western Cape Province of
C Mast et al.
Journal of physiology and pharmacology : an official journal of the Polish Physiological Society, 65(5), 623-631 (2014-11-06)
The use of glutathione (GSH) and sulfate for the detoxification of paracetamol (acetaminophen, APAP) could occur at the expense of the physiological uses of cysteine (Cys). Indeed GSH and sulfate both originate from Cys. Significant APAP-induced Cys loss could generate
Extrasynaptic glutamate release through cystine/glutamate antiporter contributes to ischemic damage.
Federico N Soria et al.
The Journal of clinical investigation, 124(8), 3645-3655 (2014-07-19)
During brain ischemia, an excessive release of glutamate triggers neuronal death through the overactivation of NMDA receptors (NMDARs); however, the underlying pathways that alter glutamate homeostasis and whether synaptic or extrasynaptic sites are responsible for excess glutamate remain controversial. Here
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