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About This Item
Linear Formula:
CH3SO2Na
CAS Number:
Molecular Weight:
102.09
NACRES:
NA.22
PubChem Substance ID:
UNSPSC Code:
12352100
EC Number:
243-669-6
MDL number:
Assay:
85%
InChI key
LYPGDCWPTHTUDO-UHFFFAOYSA-M
InChI
1S/CH4O2S.Na/c1-4(2)3;/h1H3,(H,2,3);/q;+1/p-1
SMILES string
[Na+].CS([O-])=O
grade
technical grade
assay
85%
mp
222-226 °C (dec.) (lit.)
functional group
sulfinic acid
Quality Level
Related Categories
General description
Sodium methanesulfinate is an aliphatic sodium sulfinate. Conjugate addition of sodium methanesulfinate to vinyl heterocycles has been described. Cross-coupling reaction between aryl boronic acid and sodium methanesulfinate has been studied. Its stock solution was prepared from methanesulfonic acid by adding one equivalent of sodium hydroxide and diluting it to 4M.
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A mild and efficient new synthesis of aryl sulfones from boronic acids and sulfinic acid salts.
Beaulieu C, et al.
Tetrahedron Letters, 45(16), 3233-3236 (2004)
Design, Synthesis and Discovery of 1-(2-(6-Chloro-3-methylsulfonyl)-naphthyl)-1H-pyrazole-5-carboxylamides as Highly Potent Factor Xa Inhibitors.
Jia ZJ, et al.
Chemical & Pharmaceutical Bulletin, 57(9), 1004-1007 (2009)
R Bruck et al.
Journal of hepatology, 35(4), 457-464 (2001-10-30)
Reactive oxygen species and oxidative stress were implicated in hepatic stellate cell activation and liver fibrosis. The aim of the present study was to examine whether the administration of free radical scavengers in vivo would prevent experimentally-induced hepatic cirrhosis in
Matthew L Dawson et al.
Proceedings of the National Academy of Sciences of the United States of America, 109(46), 18719-18724 (2012-10-24)
Airborne particles affect human health and significantly influence visibility and climate. A major fraction of these particles result from the reactions of gaseous precursors to generate low-volatility products such as sulfuric acid and high-molecular weight organics that nucleate to form
C F Babbs et al.
Free radical biology & medicine, 6(5), 493-503 (1989-01-01)
A major impediment to the confirmation of free radical mechanisms in pathogenesis is a lack of direct, chemical evidence that oxygen centered free radicals actually arise in living tissues in quantities sufficient to cause serious damage. This investigation was conducted
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