Skip to Content
Merck
CN

740950

Solketal methacrylate

50 wt. % in dichloromethane, contains ~280 ppm 4-tert-butylcatechol as inhibitor

Synonym(s):

(2,2-Dimethyl-1,3-dioxolan-4-yl)methyl methacrylate, GMAK, Glycerine-1-methacrylate acetone ketal, SMA

Sign In to View Organizational & Contract Pricing.

Select a Size

About This Item

Empirical Formula (Hill Notation):
C10H16O4
CAS Number:
7098-80-8
Molecular Weight:
200.23
NACRES:
NA.23
PubChem Substance ID:
329765454
UNSPSC Code:
12162002
MDL number:
MFCD21334174

Key Documents

View All Documentation
Technical Service
Need help? Our team of experienced scientists is here for you.
Let Us Assist
Technical Service
Need help? Our team of experienced scientists is here for you.
Let Us Assist

InChI

1S/C10H16O4/c1-7(2)9(11)12-5-8-6-13-10(3,4)14-8/h8H,1,5-6H2,2-4H3

SMILES string

CC(=C)C(=O)OCC1COC(C)(C)O1

InChI key

JPFPDGRVRGETED-UHFFFAOYSA-N

form

liquid

contains

~280 ppm 4-tert-butylcatechol as inhibitor

concentration

50 wt. % in dichloromethane

refractive index

n20/D 1.438

density

1.153 g/mL at 25 °C

storage temp.

−20°C

signalword

Danger

Hazard Classifications

Carc. 2 - Eye Dam. 1 - Flam. Liq. 3 - Skin Irrit. 2 - STOT SE 3

target_organs

Central nervous system, Respiratory system

Storage Class

3 - Flammable liquids

wgk

WGK 2

flash_point_f

81.0 °F

flash_point_c

27.2 °C

Regulatory Information

新产品
This item has

Choose from one of the most recent versions:

Certificates of Analysis (COA)

Lot/Batch Number
MKBH7260V

Don't see the Right Version?

If you require a particular version, you can look up a specific certificate by the Lot or Batch number.

Search for a COA

Already Own This Product?

Find documentation for the products that you have recently purchased in the Document Library.

Visit the Document Library

Caroline J Zeiss et al.
G3 (Bethesda, Md.), 9(8), 2637-2646 (2019-07-03)
Anthracyclines cause progressive cardiotoxicity whose ultimate severity is individual to the patient. Genetic determinants contributing to this variation are difficult to study using current mouse models. Our objective was to determine whether a spectrum of anthracycline induced cardiac disease can
Hiroyasu Okazaki et al.
Experimental lung research, 45(7), 188-199 (2019-07-13)
Purpose/Aim of the Study: Wnt/β-catenin signaling was reported to be activated in pulmonary fibrosis, and was focused on as a target for antifibrotic therapy. However, the mechanism how the inhibition of Wnt/β-catenin signaling ameliorate pulmonary fibrosis has not been fully
Justin A Bishop et al.
The American journal of surgical pathology, 43(8), 1023-1032 (2019-05-17)
Salivary gland adenocarcinoma not otherwise specified (NOS) is a heterogenous group, likely containing distinct tumors not yet characterized. A growing number of low to intermediate-grade salivary carcinomas are now known to harbor tumor-specific gene fusions. On occasion, identifying a novel
Sarah L O'Beirne et al.
Journal of immunology (Baltimore, Md. : 1950), 195(6), 2788-2796 (2015-08-14)
Epithelial to mesenchymal cell transition (EMT), whereby fully differentiated epithelial cells transition to a mesenchymal phenotype, has been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF). CXCR3 and its ligands are recognized to play a protective role in pulmonary
Rui Zheng et al.
The Journal of investigative dermatology, 139(9), 2029-2038 (2019-03-18)
After cutaneous injury, keratinocytes secrete paracrine factors that regulate wound cell functions; dysregulation of this signaling can lead to wound pathologies. Previously, we established that keratinocyte integrin α3β1 promotes wound angiogenesis through paracrine stimulation of endothelial cells. We hypothesize here
View All Related Papers

Our team of scientists has experience in all areas of research including Life Science, Material Science, Chemical Synthesis, Chromatography, Analytical and many others.

Contact Technical Service