A66707
4-Amino-1,8-naphthalimide
96%
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About This Item
Empirical Formula (Hill Notation):
C12H8N2O2
CAS Number:
Molecular Weight:
212.20
MDL number:
PubChem Substance ID:
Assay
96%
density
1.105 g/mL at 25 °C (lit.)
SMILES string
Nc1ccc2C(=O)NC(=O)c3cccc1c23
InChI
1S/C12H8N2O2/c13-9-5-4-8-10-6(9)2-1-3-7(10)11(15)14-12(8)16/h1-5H,13H2,(H,14,15,16)
InChI key
SSMIFVHARFVINF-UHFFFAOYSA-N
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Jun Feng Zhang et al.
Organic letters, 13(5), 1190-1193 (2011-02-09)
A naphthalimide-based highly selective colorimetric and ratiometric fluorescent probe for the fluoride ion displayed both one- and two-photon ratiometric changes. Upon reaction with the F(-) (TBA(+) and Na(+) salts) anion in CH(3)CN as well as in aqueous buffer solution, probe
Emma B Veale et al.
The Journal of organic chemistry, 75(16), 5513-5525 (2010-08-14)
The synthesis and characterization of three bis-1,8-naphthalimide-containing Tröger's bases 1-3, formed from the corresponding 4-amino-1,8-naphthalimide precursors 7-9 in a single step, is described. The photophysical investigation of 1-3 and 7-9 was carried out in various organic solvents as well as
Marco A Alcala et al.
Nanomedicine : nanotechnology, biology, and medicine, 7(3), 249-258 (2010-10-16)
Surgery is currently the best approach for treating either primary or metastatic hepatic malignancies. Because only 20% of hepatic cancers are operable in patients, several types of regional therapy (RT) are emerging as alternate treatment modalities. However, RTs can have
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Nucleic acids research, 36(13), 4454-4464 (2008-07-08)
The consequences of PARP-1 disruption or inhibition on DNA single-strand break repair (SSBR) and radio-induced lethality were determined in synchronized, isogenic HeLa cells stably silenced or not for poly(ADP-ribose) polymerase-1 (PARP-1) (PARP-1(KD)) or XRCC1 (XRCC1(KD)). PARP-1 inhibition prevented XRCC1-YFP recruitment
S García et al.
Annals of the rheumatic diseases, 67(5), 631-637 (2007-09-25)
To investigate the effect of poly(ADP-ribose) polymerase (PARP) inhibition on the production of inflammatory mediators and proliferation in tumour necrosis factor (TNF)-stimulated fibroblast-like synoviocytes (FLS) from patients with rheumatoid arthritis (RA). Cultured FLS from patients with RA were treated with
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