G11608
Guanidineacetic acid
99%
Synonym(s):
N-Amidinoglycine, N-Guanylglycine, Glycocyamine
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About This Item
Linear Formula:
H2NC(=NH)NHCH2CO2H
CAS Number:
Molecular Weight:
117.11
Beilstein:
1759179
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22
Quality Level
Assay
99%
mp
300 °C (lit.)
SMILES string
NC(=N)NCC(O)=O
InChI
1S/C3H7N3O2/c4-3(5)6-1-2(7)8/h1H2,(H,7,8)(H4,4,5,6)
InChI key
BPMFZUMJYQTVII-UHFFFAOYSA-N
Related Categories
Storage Class Code
11 - Combustible Solids
WGK
WGK 1
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
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J Michiels et al.
Poultry science, 91(2), 402-412 (2012-01-19)
Creatine, (CREA) a central constituent in energy metabolism, is obtained from dietary animal protein or de novo synthesis from guanidinoacetic acid (GAA). Especially in all-vegetable diets, supplemental CREA or GAA may restore the CREA availability in tissues, and hence, improve
Saadet Mercimek-Mahmutoglu et al.
Molecular genetics and metabolism, 105(1), 155-158 (2011-10-25)
A 4-year-old female with history of developmental regression and autistic features was diagnosed with guanidinoacetate methyltransferase deficiency at age 21 months. Upon treatment, she showed improvements in her developmental milestones, sensorial-neural hearing loss and brain atrophy on cranial-MRI. The creatine/choline
Stefan Pilz et al.
Heart (British Cardiac Society), 97(15), 1222-1227 (2011-05-12)
Homoarginine is an amino acid which may play a role in nitric oxide and energy metabolism. Low homoarginine concentrations have previously been linked to increased mortality, but the underlying mechanisms remain unknown. To investigate whether serum homoarginine concentrations are associated
Joseph D T Ndika et al.
Molecular genetics and metabolism, 106(1), 48-54 (2012-03-06)
Arginine:glycineamidinotransferase (AGAT/GATM) deficiency has been described in 9 patients across 4 families. Here we describe the clinical outcome and response to creatine supplementation in a patient of the second family affected with AGAT deficiency-a 9-year-old girl. Delayed motor milestones were
David Cheillan et al.
Orphanet journal of rare diseases, 7, 96-96 (2012-12-14)
A population of patients with unexplained neurological symptoms from six major French university hospitals was screened over a 28-month period for primary creatine disorder (PCD). Urine guanidinoacetate (GAA) and creatine:creatinine ratios were measured in a cohort of 6,353 subjects to
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