05-5680
Copper
SAJ first grade, ≥99.5%
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About This Item
Empirical Formula (Hill Notation):
Cu
CAS Number:
Molecular Weight:
63.55
UNSPSC Code:
12141711
PubChem Substance ID:
EC Number:
231-159-6
MDL number:
InChI key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
InChI
1S/Cu
SMILES string
[Cu]
grade
SAJ first grade
assay
≥99.5%
form
turnings
reaction suitability
reagent type: catalyst
core: copper
availability
available only in Japan
resistivity
1.673 μΩ-cm, 20°C
bp
2567 °C (lit.)
mp
1083.4 °C (lit.)
density
8.94 g/mL at 25 °C (lit.)
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type P3 (EN 143) respirator cartridges
signalword
Warning
hcodes
pcodes
Hazard Classifications
Aquatic Acute 1 - Aquatic Chronic 1
Storage Class
11 - Combustible Solids
Regulatory Information
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Stephen G Kaler et al.
The New England journal of medicine, 358(6), 605-614 (2008-02-08)
Menkes disease is a fatal neurodegenerative disorder of infancy caused by diverse mutations in a copper-transport gene, ATP7A. Early treatment with copper injections may prevent death and illness, but presymptomatic detection is hindered by the inadequate sensitivity and specificity of
Daniel L Priebbenow et al.
Organic letters, 15(24), 6155-6157 (2013-11-28)
A method has been developed for the preparation of N-alkynylated sulfoximines involving the copper-catalyzed decarboxylative coupling of sulfoximines with aryl propiolic acids. A range of substituents on both the sulfoximidoyl moiety and the aryl group of the propiolic acid were
Hiroshi Sato et al.
Science (New York, N.Y.), 343(6167), 167-170 (2013-12-18)
Carbon monoxide (CO) produced in many large-scale industrial oxidation processes is difficult to separate from nitrogen (N2), and afterward, CO is further oxidized to carbon dioxide. Here, we report a soft nanoporous crystalline material that selectively adsorbs CO with adaptable
Magnus Andersson et al.
Nature structural & molecular biology, 21(1), 43-48 (2013-12-10)
Heavy metals in cells are typically regulated by PIB-type ATPases. The first structure of the class, a Cu(+)-ATPase from Legionella pneumophila (LpCopA), outlined a copper transport pathway across the membrane, which was inferred to be occluded. Here we show by
Yan Meng et al.
Biochimica et biophysica acta, 1690(3), 208-219 (2004-10-30)
Hepatic abnormalities in Long-Evans Cinnamon (LEC) rats, an animal model of Wilson disease (WD), were restored by the expression of the human ATP7B cDNA under the control of CAG promoter. Expression of ATP7B transcript and protein in the liver of
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