InChI
1S/2Na.4O.W/q2*+1;;;2*-1;
SMILES string
[Na+].[Na+].[O-][W]([O-])(=O)=O
InChI key
XMVONEAAOPAGAO-UHFFFAOYSA-N
grade
analytical standard
shelf life
limited shelf life, expiry date on the label
packaging
ampule of
solubility
H2O: soluble
Preparation Note
prepared with Na2WO4, NaOH and H2O
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Met. Corr. 1 - Skin Irrit. 2
Storage Class
8B - Non-combustible corrosive hazardous materials
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Regulatory Information
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M Milagros Gómez-Gómez et al.
Talanta, 84(4), 1011-1018 (2011-05-03)
It is known that oral administration of sodium tungstate preserves the pancreatic beta cell function in diabetic rats. Healthy and streptozotocin-induced diabetic rats were treated with sodium tungstate for one, three or six weeks, after which the species of W
C J Rodriguez-Hernandez et al.
FEBS letters, 586(3), 270-276 (2012-01-17)
Tungstate counteracts diabetes and obesity in animal models, but its molecular mechanisms remain elusive. Our Saccharomyces cerevisiae-based approach has found that tungstate alleviated the growth defect induced by nutrient stress and enhanced the activation of the GCN pathway. Tungstate relieved
C J Rodriguez-Hernandez et al.
FEBS letters, 587(10), 1579-1586 (2013-04-17)
Both radiotherapy and most effective chemotherapeutic agents induce different types of DNA damage. Here we show that tungstate modulates cell response to DNA damaging agents. Cells treated with tungstate were more sensitive to etoposide, phleomycin and ionizing radiation (IR), all
Mustafa Aydemir et al.
Biological trace element research, 148(2), 216-223 (2012-02-22)
Recently, sodium tungstate was suggested to improve cardiac performance of diabetic rats in perfused hearts based on its insulinomimetic activity. In this study, we aimed to investigate the cellular and molecular mechanisms underlying this beneficial effect of sodium tungstate. Tungstate
Cynthia D Fastje et al.
Chemico-biological interactions, 196(3), 89-95 (2011-05-14)
The etiology of childhood leukemia is not known. Strong evidence indicates that precursor B-cell Acute Lymphoblastic Leukemia (Pre-B ALL) is a genetic disease originating in utero. Environmental exposures in two concurrent, childhood leukemia clusters have been profiled and compared with
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