InChI
1S/2Na.4O.W/q2*+1;;;2*-1;
SMILES string
[Na+].[Na+].[O-][W]([O-])(=O)=O
InChI key
XMVONEAAOPAGAO-UHFFFAOYSA-N
grade
analytical standard
shelf life
limited shelf life, expiry date on the label
packaging
ampule of
solubility
H2O: soluble
Preparation Note
prepared with Na2WO4, NaOH and H2O
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Met. Corr. 1 - Skin Irrit. 2
Storage Class
8B - Non-combustible corrosive hazardous materials
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Regulatory Information
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Marta Amigó-Correig et al.
PloS one, 7(7), e39087-e39087 (2012-07-18)
This study aims at exploring the effects of sodium tungstate treatment on hypothalamic plasticity, which is known to have an important role in the control of energy metabolism. Adult lean and high-fat diet-induced obese mice were orally treated with sodium
Josie E Bamford et al.
Environmental toxicology and chemistry, 30(10), 2312-2318 (2011-08-02)
Due to unknown effects of the potential exposure of the terrestrial environment to tungsten substances, a series of toxicity studies of sodium tungstate (Na(2) WO(4) ) was conducted. The effect on earthworm (Eisenia fetida) survival and reproduction was examined using
M Milagros Gómez-Gómez et al.
Talanta, 84(4), 1011-1018 (2011-05-03)
It is known that oral administration of sodium tungstate preserves the pancreatic beta cell function in diabetic rats. Healthy and streptozotocin-induced diabetic rats were treated with sodium tungstate for one, three or six weeks, after which the species of W
M Amigó-Correig et al.
Diabetes, obesity & metabolism, 13(3), 235-242 (2011-01-06)
Sodium tungstate is an anti-obesity drug targeting peripheral tissues. In vivo, sodium tungstate reduces body weight gain and food intake through increasing energy expenditure and lipid oxidation, but it also modulates hypothalamic gene expression when orally administered, raising the possibility
Delia Zafra et al.
FEBS letters, 587(3), 291-296 (2012-12-25)
Tungstate treatment ameliorates experimental diabetes by increasing liver glycogen deposition through an as yet unidentified mechanism. The signalling mechanism of tungstate was studied in CHOIR cells and primary cultured hepatocytes. This compound exerted its pro-glycogenic effects through a new G-protein-dependent
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