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Merck
CN

18951

1-Naphthyl isothiocyanate

derivatization grade (HPLC), LiChropur™, ≥98.5% (HPLC and GC)

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About This Item

Linear Formula:
C10H7NCS
CAS Number:
551-06-4
Molecular Weight:
185.24
NACRES:
NA.22
PubChem Substance ID:
329751761
UNSPSC Code:
41116105
EC Number:
208-990-8
MDL number:
MFCD00003882
Assay:
≥98.5% (HPLC and GC)
Form:
solid

Key Documents

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InChI key

JBDOSUUXMYMWQH-UHFFFAOYSA-N

InChI

1S/C11H7NS/c13-8-12-11-7-3-5-9-4-1-2-6-10(9)11/h1-7H

SMILES string

S=C=Nc1cccc2ccccc12

grade

derivatization grade (HPLC)

assay

≥98.5% (HPLC and GC)

form

solid

quality

LiChropur™

technique(s)

HPLC: suitable

mp

55.5-57 °C (lit.)

Quality Level

100

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Other Notes

Discover LiChropur reagents ideal for HPLC or LC-MS analysis

Legal Information

LiChropur is a trademark of Merck KGaA, Darmstadt, Germany

pictograms

Skull and crossbonesHealth hazard
GHS06,GHS08

signalword

Danger

Hazard Classifications

Acute Tox. 3 Oral - Eye Irrit. 2 - Resp. Sens. 1 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

Storage Class

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

危险化学品
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Certificates of Analysis (COA)

Lot/Batch Number
BCCG7719
BCCF4405
BCBW8813
BCBW7587
BCBW0368

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N Mori et al.
Die Pharmazie, 65(7), 457-460 (2010-07-29)
The intestinal absorption of mizoribine, an imidazole nucleoside, is mediated by concentrative nucleoside transporter (CNT)1 and CNT2 in rat. Previously, bile and bile salts such as sodium glycocholate were found to suppress the intestinal absorption of mizoribine. In the present
Matthew Quinn et al.
American journal of physiology. Gastrointestinal and liver physiology, 302(1), G182-G193 (2011-10-08)
Cholestatic patients often present with clinical features suggestive of adrenal insufficiency. In the bile duct-ligated (BDL) model of cholestasis, the hypothalamic-pituitary-adrenal (HPA) axis is suppressed. The consequences of this suppression on cholangiocyte proliferation are unknown. We evaluated 1) HPA axis
Igor Sukhotnik et al.
Pediatric surgery international, 28(2), 161-169 (2011-10-13)
Progressive hyperbilirubinemia and end-stage liver failure are among the most serious complications of short bowel syndrome (SBS), representing the principle cause of death in a majority of fatal cases. In the current study, we examined the effects of alpha-naphthylisothiocyanate (ANIT)-induced
Fan Yang et al.
Journal of ethnopharmacology, 144(3), 599-604 (2012-10-13)
To evaluate the hepatoprotective effect of the root and rhizome of Rhubarb (Rhei Radix et Rhizoma) against α-naphthylisothiocyanate (ANIT)-induced liver injury using metabolic profile of bile acids (BAs) along with biochemical parameters and histological alterations. Ultra-performance liquid chromatography coupled with
James P Luyendyk et al.
Toxicological sciences : an official journal of the Society of Toxicology, 119(1), 233-243 (2010-10-27)
Alpha-naphthylisothiocyanate (ANIT)-induced cholestatic liver injury causes tissue factor (TF)-dependent coagulation in mice, and TF deficiency reduces ANIT-induced liver injury. However, the mechanism whereby TF contributes to hepatotoxicity in this model is not known. Utilizing pharmacological and genetic strategies, we evaluated
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