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Merck
CN

57412

Indophenol

Synonym(s):

Phenolindophenol

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About This Item

Empirical Formula (Hill Notation):
C12H9NO2
CAS Number:
Molecular Weight:
199.21
EC Number:
207-913-5
UNSPSC Code:
12171500
PubChem Substance ID:
Beilstein/REAXYS Number:
2095656
MDL number:
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InChI key

RSAZYXZUJROYKR-UHFFFAOYSA-N

InChI

1S/C12H9NO2/c14-11-5-1-9(2-6-11)13-10-3-7-12(15)8-4-10/h1-8,14H

SMILES string

Oc1ccc(cc1)N=C2C=CC(=O)C=C2

mp

>300 °C (lit.)

suitability

suitable for microscopy

Regulatory Information

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C Bishop et al.
Neuroscience, 121(3), 649-657 (2003-10-22)
Loss of dopaminergic innervation to the striatum increases the sensitivity of dopamine (DA) D1 and serotonin (5-HT) 5-HT2 receptor signaling. Previous work from our laboratory has shown that systemic co-administration of D1 and 5-HT2 receptor agonists leads to the synergistic
Jiasheng Chen et al.
The Journal of urology, 189(5), 1982-1988 (2012-11-20)
We examined the effects of the 5-HT2A/2C receptor agonist DOI on micturition in chronic spinal cord injured rats. Female Sprague-Dawley® rats were used. Spinal cord injury was produced by transection at the T10 level. A cystometric study was performed 8
Bríd Aine Nic Dhonnchadha et al.
Behavioural brain research, 147(1-2), 175-184 (2003-12-09)
DOI [(+/-)-1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane] displays a high affinity for the rat 5-HT2A, 5-HT2B and 5-HT2C receptors (pKi 7.3, 7.4 and 7.8, respectively) and acts as an agonist. DOI (0.5-4 mg/kg, i.p. 30 min pre-test) increased the number of punished passages in the
M Hassanain et al.
Brain research, 981(1-2), 201-209 (2003-07-30)
Previous studies have established that the expression of defensive rage behavior in the cat is mediated over reciprocal pathways that link the medial hypothalamus and the dorsolateral quadrant of the midbrain periaqueductal gray matter (PAG). The present study was designed
Christen M Cormier et al.
Journal of neurotrauma, 27(10), 1805-1818 (2010-08-12)
Autonomic dysreflexia consistently develops in patients and in rats after severe upper thoracic spinal cord injury (SCI) as a result of exaggerated spinal sympathetic excitation. In this study we induced episodic hypertension in rats after varying degrees of SCI severity

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