75690
Oxalic acid
purum, anhydrous, ≥97.0% (RT)
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About This Item
Linear Formula:
HO2CCO2H
CAS Number:
Molecular Weight:
90.03
EC Number:
205-634-3
UNSPSC Code:
12352100
PubChem Substance ID:
Beilstein/REAXYS Number:
385686
MDL number:
InChI key
MUBZPKHOEPUJKR-UHFFFAOYSA-N
InChI
1S/C2H2O4/c3-1(4)2(5)6/h(H,3,4)(H,5,6)
SMILES string
OC(=O)C(O)=O
vapor density
4.4 (vs air)
vapor pressure
<0.01 mmHg ( 20 °C)
grade
purum
assay
≥97.0% (RT)
impurities
~1% water
mp
189.5 °C (dec.)
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signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 4 Dermal - Acute Tox. 4 Oral - Eye Dam. 1
Storage Class
11 - Combustible Solids
wgk
WGK 1
ppe
dust mask type N95 (US), Eyeshields, Gloves
Regulatory Information
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G M Gadd
Advances in microbial physiology, 41, 47-92 (1999-09-29)
The production of organic acids by fungi has profound implications for metal speciation, physiology and biogeochemical cycles. Biosynthesis of oxalic acid from glucose occurs by hydrolysis of oxaloacetate to oxalate and acetate catalysed by cytosolic oxaloacetase, whereas on citric acid
L K Massey et al.
Journal of agricultural and food chemistry, 49(9), 4262-4266 (2001-09-18)
Consumption of soybeans and food products made from them is increasing because of their desirable nutritional value. However, the oxalate content of seeds from 11 cultivars of soybean showed relatively high levels of total oxalate from 0.67 to 3.5 g/100
R de Water et al.
American journal of kidney diseases : the official journal of the National Kidney Foundation, 33(4), 761-771 (1999-04-09)
Urinary calcium oxalate (CaOx) crystals and crystal agglomerates are normally harmlessly excreted, but in nephrolithiasis they are retained by tubular epithelial cells and shifted into the renal interstitium. This crystalline material induces an inflammatory response consisting of an increase in
R Shapiro et al.
Transplantation, 72(3), 428-432 (2001-08-15)
The appropriate use of liver transplantation in children with type-1 primary hyperoxaluria (PH-1) is not well established. We reviewed our experience with 36 children with PH-1, including 12 who underwent liver transplantation. From 1989-1998, 36 children from 10 families in
A Amoroso et al.
Journal of the American Society of Nephrology : JASN, 12(10), 2072-2079 (2001-09-20)
Primary hyperoxaluria type 1 (PH1) is an autosomal recessive disorder that is caused by a deficiency of alanine: glyoxylate aminotransferase (AGT), which is encoded by a single copy gene (AGXT). Molecular diagnosis was used in conjunction with clinical, biochemical, and
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