95000
1,1-Dichloroethylene
puriss., ≥99.5% (GC)
Synonym(s):
Vinylidene chloride
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About This Item
Linear Formula:
CH2=CCl2
CAS Number:
Molecular Weight:
96.94
EC Number:
200-864-0
UNSPSC Code:
12162002
MDL number:
Beilstein/REAXYS Number:
1733365
vapor density
3.46 (vs air)
vapor pressure
9.68 psi ( 20 °C)
grade
puriss.
assay
≥99.5% (GC)
autoignition temp.
968 °F
contains
~0.02% 4-methoxyphenol as stabilizer
expl. lim.
15.5 %
refractive index
n20/D 1.426
bp
30-32 °C
mp
−122 °C (lit.)
density
1.213 g/mL at 20 °C (lit.)
storage temp.
2-8°C
SMILES string
ClC(Cl)=C
InChI
1S/C2H2Cl2/c1-2(3)4/h1H2
InChI key
LGXVIGDEPROXKC-UHFFFAOYSA-N
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Danger
Hazard Classifications
Acute Tox. 3 Oral - Acute Tox. 4 Inhalation - Aquatic Chronic 2 - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 1 - STOT RE 1 Inhalation - STOT RE 2 Oral
target_organs
Liver, Nose
Storage Class
3 - Flammable liquids
wgk
WGK 3
flash_point_f
-9.4 °F - closed cup
flash_point_c
-23.0 °C - closed cup
ppe
Eyeshields, Faceshields, Gloves
Regulatory Information
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Young Kim et al.
Biotechnology and bioengineering, 80(5), 498-508 (2002-10-02)
Batch kinetic and inhibition studies were performed for the aerobic cometabolism of 1,1,1-trichloroethane (1,1,1-TCA), 1,1-dichloroethylene (1,1-DCE), and 1,1-dichloroethane (1,1-DCA) by a butane-grown mixed culture. These chlorinated aliphatic hydrocarbons (CAHs) are often found together as cocontaminants in groundwater. The maximum degradation
Masarin Ban et al.
Toxicology, 226(2-3), 161-171 (2006-08-02)
Occupational allergy and asthma is a challenging issue in the developing countries. Chemicals inhaled in the workplaces may act not only as allergens but also as immune response modifiers, contributing to asthma exacerbation. In this study, we tested the adjuvant
Erik J Martin et al.
The Journal of pharmacology and experimental therapeutics, 313(1), 95-103 (2005-01-01)
1,1-Dichloroethylene (DCE) causes pulmonary injury that is characterized by necrosis of bronchiolar Clara cells. Mitochondria have been identified as an early target in the toxic response. Because mitochondria have been implicated in both necrotic and apoptotic cell death, we have