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Merck
CN

B100

Sigma-Aldrich

Nucleosides

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EC Number:
UNSPSC Code:
41106305
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storage temp.

2-8°C

Other Notes

4 Nucleosides, 100mg of each
A9251Adenosine
C4654 Cytidine free base, cell culture tested
G6752Guanosine
U3750Uridine

Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

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Sang Jun Lee et al.
Proceedings of the National Academy of Sciences of the United States of America, 106(46), 19515-19520 (2009-11-06)
Small molecules generally activate or inhibit gene transcription as externally added substrates or as internally accumulated end-products, respectively. Rarely has a connection been made that links an intracellular intermediary metabolite as a signal of gene expression. We report that a
Ling Liu et al.
Microbiology and immunology, 59(7), 419-425 (2015-06-23)
We have previously demonstrated that high-density lipoprotein (HDL) can specifically bind to streptococcal collagen-like protein 1 (Scl1) of M41-type group A Streptococcus (GAS). However, the pathological or physiological significance of Scl1-HDL interaction is unknown. Here, the hypothesis that HDL acts
Thanaporn Liangsupree et al.
Scientific reports, 9(1), 11235-11235 (2019-08-04)
Low-density lipoprotein (LDL) is considered the major risk factor for the development of atherosclerotic cardiovascular diseases (ASCVDs). A novel and rapid method for the isolation of LDL from human plasma was developed utilising affinity chromatography with monolithic stationary supports. The
Richard S Finkel et al.
PloS one, 7(4), e35462-e35462 (2012-05-05)
Spinal Muscular Atrophy (SMA) is a neurodegenerative motor neuron disorder resulting from a homozygous mutation of the survival of motor neuron 1 (SMN1) gene. The gene product, SMN protein, functions in RNA biosynthesis in all tissues. In humans, a nearly
Bettina Mittendorfer et al.
The Journal of clinical endocrinology and metabolism, 101(11), 4151-4160 (2016-11-05)
High-plasma very low-density lipoprotein (VLDL) triglyceride (TG) concentration and alterations in VLDL-TG metabolism are associated with cardiometabolic disease. This study sought to evaluate the interrelationships among factors purported to regulate VLDL-TG metabolism in a large cohort of men and women

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