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Merck
CN

C0297

Sigma-Aldrich

Monoclonal Anti-Caldesmon antibody produced in mouse

clone C21, ascites fluid

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About This Item

MDL number:
MFCD00164554
UNSPSC Code:
12352203

Key Documents

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biological source

mouse

conjugate

unconjugated

antibody form

ascites fluid

antibody product type

primary antibodies

clone

C21, monoclonal

contains

15 mM sodium azide

species reactivity

mammals

technique(s)

indirect ELISA: 1:2,500
western blot: suitable

isotype

IgG1

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... CALD1(800)
mouse ... Cald1(109624)
rat ... Cald1(25687)

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Immunogen

chicken caldesmon

Biochem/physiol Actions

The antibody is directed against an epitope near the carboxy-terminus, Ca2+/calmodulin and F-actin binding site of caldesmon. Using ELISA, the antibody is able to inhibit caldesmon binding to Ca2+/calmodulin and F-actin. By immunoblotting, the antibody shows broad species reactivity to non-muscle caldesmon of mammalian cells.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number
127K4783
097K4811
085K4802
021K4840

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J J Lin et al.
Hybridoma, 7(3), 273-288 (1988-06-01)
Monoclonal antibodies, C2, C9, C18, C21 and C23, against chicken gizzard caldesmon have been prepared and characterized. These antibodies reacted with gizzard caldesmon (150 KDa) by enzyme-linked immunosorbent assay and protein immunoblotting. Immunofluorescence microscopy with these antibodies on cultured gizzard
Julie L Boerner et al.
Oncogene, 22(43), 6679-6689 (2003-10-14)
Mutations within members of the EGF/ErbB receptor family frequently release the oncogenic potential of these receptors, resulting in the activation of downstream signaling events independent of ligand regulatory constraints. We previously have demonstrated that the signal transduction events originating from
Makoto Hosoya et al.
BioMed research international, 2016, 1781894-1781894 (2016-07-13)
Deafness is one of the most common types of congenital impairments, and at least half of the cases are caused by hereditary mutations. Mutations of the gene KIAA1199 are associated with progressive hearing loss. Its expression is abundant in human
Erik Y Zhang et al.
The American journal of pathology, 164(2), 601-612 (2004-01-27)
Partial bladder outlet obstruction (PBOO) induces remodeling of urinary bladder smooth muscle (detrusor). We demonstrate an increase in bladder wall mass, muscle bundle size, and a threefold increase in the cross-sectional area of detrusor myocytes following PBOO in male New
M J McManus et al.
Proceedings of the National Academy of Sciences of the United States of America, 94(21), 11351-11356 (1997-10-23)
The avian erythroblastosis viral oncogene (v-erbB) encodes a receptor tyrosine kinase that possesses sarcomagenic and leukemogenic potential. We have expressed transforming and nontransforming mutants of v-erbB in fibroblasts to detect transformation-associated signal transduction events. Coimmunoprecipitation and affinity chromatography have been
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