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C3392

Monoclonal Anti-Caspase 4 antibody produced in mouse

Name: Monoclonal Anti-Caspase 4 antibody produced in mouse
Brand: SIGMA

~2 mg/mL, clone CAS4, purified immunoglobulin, buffered aqueous solution

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About This Item

UNSPSC Code:

12352203

MDL number:

MFCD01634344

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biological source

mouse

conjugate

unconjugated

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

CAS4, monoclonal

form

buffered aqueous solution

mol wt

antigen 43 kDa

species reactivity

monkey, human

concentration

~2 mg/mL

technique(s)

indirect ELISA: suitable, microarray: suitable, western blot: 1-2 μg/mL using a whole cell extract of monkey kidney cell line COS-7

isotype

IgG1

UniProt accession no.

P49662

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... CASP4(837)

Immunogen

synthetic peptide corresponding to amino acids 119-133 of human caspase 4 protein conjugated to KLH.

Physical form

Solution in 0.01 M phosphate buffered saline, pH 7.4, containing 15 mM sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

12 - Non Combustible Liquids

wgk

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number

100K4809

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Sulindac sulfide inhibits sarcoendoplasmic reticulum Ca2+ ATPase, induces endoplasmic reticulum stress response, and exerts toxicity in glioma cells: relevant similarities to and important differences from celecoxib.

M C White et al.

Journal of neuroscience research, 91(3), 393-406 (2013-01-03)

Malignant gliomas have low survival expectations regardless of current treatments. Nonsteroidal anti-inflammatory drugs (NSAIDs) prevent cell transformation and slow cancer cell growth by mechanisms independent of cyclooxygenase (COX) inhibition. Certain NSAIDs trigger the endoplasmic reticulum stress response (ERSR), as revealed

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