C9759
Cardiotoxin from Naja mossambica mossambica
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About This Item
UNSPSC Code:
12352200
storage temp.
−20°C
Quality Level
Other Notes
Snake toxin; mixture of cardiotoxins.
Storage Class
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Regulatory Information
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Tuan H N Nguyen et al.
BMC cell biology, 11, 57-57 (2010-07-22)
In normal adult skeletal muscle, cell turnover is very slow. However, after an acute lesion or in chronic pathological conditions, such as primary myopathies, muscle stem cells, called satellite cells, are induced to proliferate, then withdraw definitively from the cell
Erika Ferrari et al.
Biosensors, 10(9) (2020-09-03)
Organs-on-chip (OoC), often referred to as microphysiological systems (MPS), are advanced in vitro tools able to replicate essential functions of human organs. Owing to their unprecedented ability to recapitulate key features of the native cellular environments, they represent promising tools
Omar Cortez-Toledo et al.
PloS one, 12(2), e0171268-e0171268 (2017-02-09)
Muscle atrophy is a prevalent condition in illness and aging. Identifying novel pathways that control muscle mass may lead to therapeutic advancement. We previously identified Nur77 as a transcriptional regulator of glycolysis in skeletal muscle. More recently, we showed that
Yuko Nitahara-Kasahara et al.
Stem cell research & therapy, 12(1), 105-105 (2021-02-06)
Multipotent mesenchymal stromal cells (MSCs) are potentially therapeutic for muscle disease because they can accumulate at the sites of injury and act as immunosuppressants. MSCs are attractive candidates for cell-based strategies that target diseases with chronic inflammation, such as Duchenne
Charlotte Debaud et al.
PloS one, 12(8), e0182454-e0182454 (2017-08-31)
We previously reported the development of a new acquired neurogenic HO (NHO) mouse model, combining spinal cord transection (SCI) and chemical muscle injury. Pathological mechanisms responsible for ectopic osteogenesis after central neurological damage are still to be elucidated. In this
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