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Merck
CN

E4269

SAFC

LONG® EGF human

powder, recombinant, expressed in E. coli, suitable for cell culture

Synonym(s):

Epidermal Growth Factor human, EGF

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About This Item

CAS Number:
MDL number:
UNSPSC Code:
12352200
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biological source

human

recombinant

expressed in E. coli

form

powder

potency

≤10 ng/mL ED50

mol wt

12,297 Da

manufacturer/tradename

Repligen Sweden AB

storage condition

avoid repeated freeze/thaw cycles

technique(s)

cell culture | mammalian: suitable

UniProt accession no.

storage temp.

2-8°C

Gene Information

human ... EGF(1950)

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Biochem/physiol Actions

A genetically engineered recombinant human analog of EGF

Preparation Note

Lyophilized from 0.1 M acetic acid.

Legal Information

LONG is a registered trademark of Repligen Sweden AB

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Regulatory Information

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Samar Elzein et al.
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Glioblastoma (GBM) is the most common primary brain tumor, accounting for approximately 40% of all central nervous system malignancies. Despite standard treatment consisting of surgical resection, radiotherapy and/or chemotherapy, the prognosis for GBM is poor; with a median survival of
E V A Raine et al.
Osteoarthritis and cartilage, 22(5), 698-705 (2014-03-04)
The TGF-β pathway plays a central role in joint development with polymorphism in TGF-β pathway genes implicated in osteoarthritis susceptibility. One association is to rs12901499, within intron 1 of SMAD3. Since rs12901499 is not in linkage disequilibrium with a non-synonymous
Sandrine Cornaz-Buros et al.
Cancer research, 74(22), 6610-6622 (2014-09-28)
Plasticity in cancer stem-like cells (CSC) may provide a key basis for cancer heterogeneity and therapeutic response. In this study, we assessed the effect of combining a drug that abrogates CSC properties with standard-of-care therapy in a Ewing sarcoma family
Husni Elbahesh et al.
Journal of virology, 88(12), 6714-6728 (2014-04-04)
Viruses modulate cellular signaling pathways at almost every step of the infection cycle. Cellular signaling pathways activated at later times of influenza infection have previously been investigated; however, early influenza virus-host cell interactions remain understudied. Focal adhesion kinase (FAK) is

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