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Merck
CN

G6414

Sigma-Aldrich

Monoclonal Anti-Glycogen Synthase Kinase-3β (GSK-3β) antibody produced in mouse

clone GSK-4B, ascites fluid

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About This Item

MDL number:
MFCD01633410
UNSPSC Code:
12352203

Key Documents

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biological source

mouse

conjugate

unconjugated

antibody form

ascites fluid

clone

GSK-4B, monoclonal

mol wt

antigen 47 kDa

contains

15 mM sodium azide

species reactivity

rabbit, human, mouse, rat

technique(s)

microarray: suitable
western blot: 1:500 using mouse brain cytosolic preparation

isotype

IgG1

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... GSK3B(2932)
mouse ... Gsk3b(56637)
rat ... Gsk3b(84027)

Immunogen

recombinant rabbit GSK-3β.

Application

Applications in which this antibody has been used successfully, and the associated peer-reviewed papers, are given below.
Western Blotting (1 paper)

Storage Class Code

12 - Non Combustible Liquids

WGK

nwg

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

新产品
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Certificates of Analysis (COA)

Lot/Batch Number
107K4837
126K4857
114K4769
019H4840

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Xinjun Zhao et al.
Therapeutics and clinical risk management, 11, 1371-1381 (2015-09-24)
Aldehyde dehydrogenase-2 (ALDH2) has a protective effect on ischemic heart disease. Here, we examined the protective effects of ALDH2 on cardiac fibrosis through modulation of the Wnt/β-catenin signaling pathway in a rat model of myocardial infarction (MI). Wistar rats were
Adam R Cole et al.
Methods in molecular biology (Clifton, N.J.), 468, 45-65 (2008-12-23)
Glycogen synthase kinase (GSK)-3 is a key signalling intermediate in the action ofWnts. This protein kinase is ubiquitously expressed and has high inherent activity but is inhibited by activation of Wnt signalling or activation of growth factor receptor tyrosine kinases
Huizhi Wang et al.
Journal of immunology (Baltimore, Md. : 1950), 186(2), 675-684 (2010-12-17)
The ability of IFN-β to induce IL-10 production from innate immune cells is important for its anti-inflammatory properties and is believed to contribute to its therapeutic value in treating multiple sclerosis patients. In this study, we identified that IFN-β stimulates

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