L5756
Lithocholic acid 3-sulfate disodium salt hydrate
Synonym(s):
3α-Hydroxy-5β-cholan-24-oic acid 3-sulfate, Sulfolithocholic acid
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About This Item
Empirical Formula (Hill Notation):
C24H38Na2O6S · xH2O
CAS Number:
Molecular Weight:
500.60 (anhydrous basis)
UNSPSC Code:
12161900
PubChem Substance ID:
MDL number:
SMILES string
[Na+].[Na+].[H]O[H].[H][C@]12CC[C@@]3([H])[C@]4([H])CC[C@H]([C@H](C)CCC([O-])=O)[C@@]4(C)CC[C@]3([H])[C@@]1(C)CC[C@H](C2)OS([O-])(=O)=O
assay
≥95% (TLC)
storage temp.
−20°C
Storage Class
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
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J D Hayes et al.
The Biochemical journal, 215(3), 581-588 (1983-12-01)
A purification scheme is described for the neutral glutathione S-transferases of rat liver. Discontinuous sodium dodecyl sulphate/polyacrylamide-gel electrophoresis revealed that one of these enzymes contains a previously unidentified subunit, which has a molecular mass of 23 000 Da and has
D G Oelberg et al.
The American journal of physiology, 247(1 Pt 1), G112-G115 (1984-07-01)
Lithocholic acid (LCA) and its sulfate (LCS) and glucuronide (LCG) derivatives are potent cholestatic agents. During the course of LCG-induced cholestasis in rats, calcium (Ca) salts of LCG precipitate in bile. To characterize the affinity of bile salts for Ca
Cholic acid binding by anionic glutathione-S-transferase from human liver cytosol.
N R Pattinson
Biochemical and biophysical research communications, 102(1), 403-410 (1981-09-16)
H Takikawa et al.
Journal of gastroenterology and hepatology, 10(4), 383-386 (1995-07-01)
The absorption of lithocholate and its sulfate and glucuronide in rat jejunum and terminal ileum was studied. Tracer amounts of radiolabelled bile acids were administered to the ligated intestinal segments, and their absorption was monitored by biliary excretion through a
H Takikawa et al.
Digestive diseases and sciences, 40(8), 1792-1797 (1995-08-01)
Biliary excretion of lithocholate-3-sulfate is markedly impaired in EHBR. To examine the mechanism of biliary lithocholate-3-sulfate excretion in EHBR, the effects of colchicine treatment, a vesicular transport inhibitor, and infusion of taurocholate and organic anions were studied in EHBR and
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