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About This Item
NACRES:
NB.22
UNSPSC Code:
41122107
Material:
flat bottom clear polystyrene wells, polystyrene
Size:
60 wells
Sterility:
non-sterile
Binding type:
non-treated surface
Feature:
lid, low profile
material
flat bottom clear polystyrene wells, polystyrene
sterility
non-sterile
feature
lid, low profile
packaging
case of 100 (internal packs of 10)
manufacturer/tradename
Nunc 439225
external L × W
84 mm × 59 mm , w/ lid
maximum volume
10 μL
size
60 wells
surface area
0.2 cm2 , total surface area (cm2)
well maximum volume
10 μm
working volume
10 μL
color
clear
suitability
suitable for (serological applications), suitable for (serotyping, microcytotoxicity and cell cloning studies)
binding type
non-treated surface
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General description
Create more space in lab freezers and inubators with the Nuncnunc® MicroWell™ MiniTrays, the optical clarity of which allows the trays to be used with inverted microscopes.
Application
- Serotyping
- Microcytotoxicity
- Cell cloning studies
Features and Benefits
- Limited adsorption of proteins
- Lids have keyed tabs for proper orientation
- Large raised alphanumeric coordinates
- Unique surface treatment to maximize sample and reagent mixing
- MiniTrays for serological applications have slightly smaller well dimensions
Legal Information
MicroWell is a trademark of Thermo Fisher Scientific or its subsidiaries
Nunc is a registered trademark of Thermo Fisher Scientific or its subsidiaries
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Mariana C C Silva et al.
The Journal of cell biology, 219(4) (2020-04-25)
Cohesin is essential for genome folding and inheritance. In somatic cells, these functions are both mediated by Scc1-cohesin, which in mitosis is released from chromosomes by Wapl and separase. In mammalian oocytes, cohesion is mediated by Rec8-cohesin. Scc1 is expressed
Iris Schäffner et al.
Neuron, 99(6), 1188-1203 (2018-09-11)
Autophagy is a conserved catabolic pathway with emerging functions in mammalian neurodevelopment and human neurodevelopmental diseases. The mechanisms controlling autophagy in neuronal development are not fully understood. Here, we found that conditional deletion of the Forkhead Box O transcription factors FoxO1
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