N0509
Neurotensin Fragment 1-8
≥97% (HPLC)
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About This Item
Empirical Formula (Hill Notation):
C46H71N13O14
CAS Number:
Molecular Weight:
1030.13
UNSPSC Code:
12352200
PubChem Substance ID:
MDL number:
Application
Used to identify antibody specificities in cross-reactivity studies.
Storage Class
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Regulatory Information
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C F Ferris et al.
Regulatory peptides, 12(2), 101-111 (1985-10-01)
The levels of neurotensin (NT) and its metabolite, the N-terminal octapeptide (NT1-8), identified by HPLC and measured by RIA, were increased in the hepatic-portal circulation of the anesthetized rat during perfusion of the small intestine with a lipid solution, while
M F Pozza et al.
European journal of pharmacology, 145(3), 341-343 (1988-01-19)
The electrophysiological effects of neurotensin (NT), its analog xenopsin, and the fragment NT-(1-8) on the activity of dopaminergic neurons in rat substantia nigra slices were compared. Xenopsin and NT produced a concentration-dependent increase in the firing rate. The mean firing
Y F Lu et al.
Brain research bulletin, 40(2), 135-141 (1996-01-01)
The effects of neurotensin (NT) on neurons in the central amygdaloid nucleus (ACe) were investigated in rat brain slice preparations by adding the peptide to the perfusing medium. Of 115 ACe neurons, 69 cells (60%) showed excitatory responses and 10
R Nustede et al.
Pancreas, 4(1), 114-119 (1989-01-01)
Stimulation of the exocrine pancreas is one of the biological effects attributed to the tridecapeptide neurotensin (NT). Only the C-terminal 8-13 fragment with its extremely short plasma half-life in the peripheral circulation was considered to be necessary for biological activity.
D E Hernandez et al.
Brain research, 301(1), 153-156 (1984-05-28)
Intracisternal (i.c.) administration of the endogenous tridecapeptide neurotensin (NT) has been previously shown to significantly reduce the incidence of cold-restraint stress (CRS)-induced gastric ulcers in rats. In this study we confirm the cytoprotective activity of central NT, and document structure-activity
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