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Merck
CN

P0268

Sigma-Aldrich

Pyridine-2-aldoxime methyl methanesulfonate

Synonym(s):

2-PAM methanesulfonate, 2-PAM methanesulfonate salt, P2S, Pralidoxime mesylate, Pralidoxime mesylate salt, Pralidoxime methanesulfonate

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About This Item

Empirical Formula (Hill Notation):
C7H9N2O · CH3O3S
CAS Number:
154-97-2
Molecular Weight:
232.26
EC Number:
205-839-8
UNSPSC Code:
12352103

Key Documents

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storage temp.

2-8°C

InChI

1S/C7H8N2O.CH4O3S/c1-9-5-3-2-4-7(9)6-8-10;1-5(2,3)4/h2-6H,1H3;1H3,(H,2,3,4)/b7-6+;

InChI key

WWZYJJGFUIAWNW-UHDJGPCESA-N

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Storage Class Code

11 - Combustible Solids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number
073F0113

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Diabetic ketoacidosis: an uncommon manifestation of pesticide poisoning.
Krishnan Swaminathan et al.
Diabetes care, 36(1), e4-e4 (2012-12-25)
A Bayir et al.
Human & experimental toxicology, 32(1), 45-52 (2012-10-13)
The aim of this study was to examine the effects of ubiquinone (CoQ10) on heart tissue and erythrocytes in acute organophosphate poisoning (AOP). A total of 20 rabbits were divided into three groups: sham (n = 8), pralidoxime (PAM) +
Misako Yorimitsu et al.
Magnetic resonance in medical sciences : MRMS : an official journal of Japan Society of Magnetic Resonance in Medicine, 11(1), 9-16 (2012-03-28)
Whole-heart 3-dimensional (3D) late-gadolinium-enhanced magnetic resonance (MR) imaging (WH-LGE) uses respiratory gating combined with acquisition of 3D data for the entire heart in a single scan, which permits reconstruction of any plane with high resolution. We investigated the optimal scan
S Pazooki et al.
The Medical journal of Malaysia, 66(4), 342-345 (2012-02-04)
With the establishment of the inadequate efficiency of atropines and oximes in reducing morbidity and mortality of patients poisoned by organophosphates, more attention is given to using other methods such as Fresh Frozen Plasma (FFP) as a bioscavenger to mop
Ying Wang et al.
Neurotoxicology, 33(2), 169-177 (2012-01-17)
The chemical warfare nerve agent, soman irreversibly inhibits acetylcholinesterase (AChE) leading to hypercholinergy and seizures which trigger glutamate toxicity and status epilepticus ultimately resulting in neuropathology and neurobehavioral deficits. The standard emergency treatment comprising of anticholinergic, AChE reactivator and anticonvulsant
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