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Merck
CN

SAB1305560

Anti-MAP Kinase- ERK1/ERK2 Antibody

mouse monoclonal, 784CT7.6.3

Synonym(s):

ERK1, MAPK3, Mitogen-activated protein kinase 3, PRKM3

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

Key Documents

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Product Name

MONOCLONAL ANTI-ERK1/2 antibody produced in mouse, clone 784CT7.6.3, IgG fraction of antiserum, buffered aqueous solution

biological source

mouse

Quality Level

100

antibody form

IgG fraction of antiserum

antibody product type

primary antibodies

clone

784CT7.6.3, monoclonal

form

buffered aqueous solution

mol wt

43136 Da

species reactivity

mouse, human

technique(s)

western blot: 1:1000

isotype

IgG2a

UniProt accession no.

P27361,P28482

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... MAPK3(5595)

Physical form

Supplied in PBS with 0.09% (W/V) sodium azide

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number
SG121029AB

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Guangchao Zhao et al.
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It is known that preconditional treatment with volatile anesthetics can induce tolerance of the brain to stroke. A previous study demonstrated that the involvement of TREK-1, a two-pore domain K+ channel, in sevoflurane preconditioning induced neuroprotection against focal cerebral ischemia
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Frontiers in genetics, 12, 660592-660592 (2021-05-29)
Neurofibromatosis type 1 (NF1) is a common genetic disorder characterized by cafe'-au-lait spots, skinfold freckles, the formation of neurofibromas, skeletal dysplasia, vascular dysplasia, and an increased risk of malignant tumors. In this study, two Chinese NF1 children troubled with bone
Tingting Zhang et al.
Molecular genetics & genomic medicine, 9(5), e1643-e1643 (2021-03-26)
Neurofibromatosis type 1 is an autosomal dominant inherited disease and caused by NF1 gene mutation. Its clinical manifestations include multiple cafe´-au lait (CAL) spots, skinfold freckling, neurofibroma, bone dysplasia, learning disabilities, and an increased risk of malignancy. Here, we reported
Davide D'Amico et al.
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 42(2), 462-472 (2016-08-19)
The inability to properly extinguish fear memories constitutes the foundation of several anxiety disorders, including panic disorder. Recent findings show that boosting prefrontal cortex synaptic plasticity potentiates fear extinction, suggesting that therapies that augment synaptic plasticity could prove useful in
Marisa Schmitt et al.
Molecular & cellular proteomics : MCP, 18(6), 1096-1109 (2019-03-21)
Targeted inhibition of mutated kinases using selective MAP kinase inhibitors in malignant melanoma often results in temporary improvement of clinical symptoms followed by rapid development of resistance. To gain insights in molecular processes that govern resistance, we performed SILAC-based quantitative
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