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SAB4200296

Anti-Tensin 3 (N-terminal region) antibody produced in rabbit

Name: Anti-Tensin 3 (N-terminal region) antibody produced in rabbit
Brand: SIGMA

~1.5 mg/mL, affinity isolated antibody

Synonym(s):

Anti-TEM6, Anti-TENS1, Anti-TNS3

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About This Item

NACRES:

NA.41

UNSPSC Code:

12352203

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biological source

rabbit

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

antigen ~150 kDa (rat and mouse), antigen ~180 kDa (human)

species reactivity

rat, human, mouse

concentration

~1.5 mg/mL

technique(s)

western blot: 1-2 μg/mL using HEK-293T cell lysates overexpressing human tensin 3., western blot: 1.5-3.0 μg/mL using lysates of NIH3T3 and NRK cells.

UniProt accession no.

Q68CZ2

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... TNS3(64759)
mouse ... TNS3(319939)

Immunogen

synthetic peptide corresponding to a sequence located within the N-terminal region of human tensin 3, conjugated to KLH. The corresponding sequence is highly conserved in mouse and rat tensin 3 (88% and 83% sequence identity, respectively).

Physical form

Solution in 0.01 M phosphate buffered saline, pH 7.4, containing 15 mM sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number

051M4816

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Dynamic interplay of two molecular switches enabled by the MEK1/2-ERK1/2 and IL-6-STAT3 signaling axes controls epithelial cell migration in response to growth factors.

Lyugao Qin et al.

The Journal of biological chemistry, 297(4), 101161-101161 (2021-09-05)

Cell migration is an essential physiological process, and aberrant migration of epithelial cells underlies many pathological conditions. However, the molecular mechanisms governing cell migration are not fully understood. We report here that growth factor-induced epithelial cell migration is critically dependent