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Merck
CN

SAB5600232

Anti-Acetyl-Histone H3 (Lys9) antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM161, purified immunoglobulin

Synonym(s):

H3K9ac, Histone H3 (acetyl K9)

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About This Item

NACRES:
NA.41
UNSPSC Code:
12352203
Species reactivity:
vertebrates
Clone:
RM161, monoclonal, recombinant monoclonal
Application:
ChIP, ELISA, ICC, IHC, WB, multiplexing
Citations:
2
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species reactivity

vertebrates

recombinant

expressed in HEK 293 cells

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

RM161, monoclonal, recombinant monoclonal

form

buffered aqueous glycerol solution

technique(s)

ChIP: 2-10 μg/mL, ELISA: 0.5-2 μg/mL, immunocytochemistry: 0.5-2 μg/mL, immunohistochemistry: 0.1—1 μg/mL, multiplexing: 0.05-0.5 μg/mL, western blot: 0.25— 1 μg/mL

isotype

IgG

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

acetylation (Lys9)

Gene Information

human ... H3C1(8350)

Immunogen

An acetyl-peptide corresponding to the Acetyl-Histone H3 (Lys9)

Biochem/physiol Actions

This antibody reacts to Histone H3 acetylated at Lysine 9 (K9ac). No cross reactivity with other acetylated Lysines in histone H3.

Physical form

Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

低风险生物材料
常规特殊物品
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Lin Xiao et al.
Clinical epigenetics, 13(1), 53-53 (2021-03-12)
Intervertebral disc degeneration (IDD) is a major cause of lower back pain. This study aimed at exploring the effects of histone deacetylase 4 (HDAC4) and its upstream and downstream signaling molecules on IDD development. A murine IDD model was established
Hang Xue et al.
American journal of physiology. Cell physiology, 319(2), C381-C391 (2020-06-04)
Several microRNAs (miRNAs or miRs) regulate cerebral ischemic injury outcomes; however, little is known about the role of miR-539-5p during cerebral ischemic injury or the postischemic state. Cerebral ischemic injury was modeled in vitro by exposing human cortical neurons to

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