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Merck
CN

SML4103

4-Hydroperoxycyclophosphamide

≥98% (HPLC)

Synonym(s):

2-[Bis(2-chloroethyl)amino]tetrahydro-2-oxido-2H-1,3,2-oxazaphosphorin-4-yl hydroperoxide, 4-Hydroperoxycyclofosfamide, 4-OOH-CY, Asta 6496, Perfosfamide

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About This Item

Empirical Formula (Hill Notation):
C7H15Cl2N2O4P
CAS Number:
39800-16-3
Molecular Weight:
293.08
MDL number:
MFCD01710226

Key Documents

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Quality Level

100

Assay

≥98% (HPLC)

form

powder

color

white to beige

solubility

DMSO: 2 mg/mL, clear

storage temp.

-10 to -25°C

Biochem/physiol Actions

Active metabolite of the nitrogen mustard cyclophosphamide.
4-Hydroperoxycyclophosphamide is the active metabolite of the nitrogen mustard cyclophosphamide. 4-Hydroperoxycyclophosphamide exhibits potent antineoplastic and immunosuppressive activities.

Signal Word

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Oral - Carc. 1B - Eye Dam. 1 - Muta. 1B - Repr. 1A - Skin Corr. 1B - Skin Sens. 1A

Storage Class Code

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
0000397581
0000397585
0000379172
0000379172
0000397585

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Xinfei Ma et al.
Life sciences, 274, 119331-119331 (2021-03-16)
Cyclophosphamide (CP) is a common therapeutic drug for cancer, but exposure to CP can cause acute hepatotoxicity. This study aimed to elucidate the protective effects of Ligustrazine (2, 3, 5, 6-tetramethylpyrazine, TMP) on hepatotoxicity induced by CP or its active
Charles Linker
Best practice & research. Clinical haematology, 20(1), 77-84 (2007-03-06)
Since 1986, the University of California San Francisco has developed novel approaches to autologous transplantation for acute myeloid leukemia (AML). Strategies have included intensive preparative regimens using busulfan and etoposide, and evolving strategies for pre-transplant consolidation and stem cell collection.
I Piccini et al.
The British journal of dermatology, 186(1), 129-141 (2021-09-09)
Permanent chemotherapy-induced alopecia (pCIA), for which preventive interventions remain limited, can manifest with scarring. While the underlying pathomechanisms of pCIA are unclear, depletion of epithelial hair follicle (HF) stem cells (eHFSCs) is likely to play a role. To explore the

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