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Merck
CN

W0267

Anti-WAVE-1 antibody produced in rabbit

~2 mg/mL, affinity isolated antibody, buffered aqueous solution

Synonym(s):

Anti-WASF1, Anti-Wiskott-Aldrich syndrome protein family member 1

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About This Item

UNSPSC Code:
12352203
MDL number:
MFCD04118689

Key Documents

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biological source

rabbit

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

antigen 75 kDa

species reactivity

mouse, human, rat

concentration

~2 mg/mL

technique(s)

indirect immunofluorescence: suitable, western blot: 0.25-0.5 μg/mL using rat and mouse brain cytosolic fraction (S1)

UniProt accession no.

Q92558

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... WASF1(8936)
mouse ... Wasf1(83767)
rat ... Wasf1(294568)

Immunogen

synthetic peptide corresponding to amino acids 182-200 located at the N-terminal region of human WAVE-1, conjugated to KLH. This sequence is identical in several species including mouse and rat WAVE-1 and not found in other WAVE isoforms.

Application

Yale Center for High Throughput Cell Biology IF-tested antibodies. Each antibody is tested by immunofluorescence against HUVEC cells using the Yale HTCB IF protocol. To learn more about us and Yale Center for High Throughput Cell Biology partnership, visit sigma.com/htcb-if.

Physical form

Solution in 0.01 M phosphate buffered saline, pH 7.4, containing 15 mM sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Certificates of Analysis (COA)

Lot/Batch Number
076K4752

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Yoko Ito et al.
American journal of human genetics, 103(1), 144-153 (2018-07-03)
Next-generation sequencing has been invaluable in the elucidation of the genetic etiology of many subtypes of intellectual disability in recent years. Here, using exome sequencing and whole-genome sequencing, we identified three de novo truncating mutations in WAS protein family member

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