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文件

1.16890

Fractogel EMD SO₃⁻ (S)

Name: Fractogel<TMSYMBOL></TMSYMBOL> EMD SO₃⁻ (S)
Brand: MM

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别名:

Fractogel^® EMD SO₃⁻ (S) 树脂

ligand

(Sulfoisobutyl)

质量水平

100
400

描述

strong cation exchanger, suspension in 20% ethanol and 150 mM NaCl (20-40 µm)

无菌性

sterile (Caustic Stable)

检测方案

≥90% (HPLC)

形式

resin

制造商/商品名称

Calbiochem^®

参数

8 bar max. pressure
80 cm/hr flow rate

基质活性基团

methacrylate

平均粒径

20-40 um μm

容量

150 mg binding capacity (lysozyme/ml of resin)

转变温度

flash point 35 °C (Does not sustain combustion.)

密度

1.43 g/cm³ at 20 °C

堆积密度

1000 kg/m³

分离技术

strong cation exchange

运输

ambient

储存温度

2-8°C

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包装

1.16882.0100: Fractogel^® EMD SO3- (S) Resin 100ml
1.16882.0010: Fractogel^® EMD SO3- (S) Resin 10ml
1.16882.0500: Fractogel^® EMD SO3- (S) Resin 500ml
1.16882.5000: Fractogel^® EMD SO3- (S) Resin 5L

警告

Toxicity: Standard Handling (A)

分析说明

Appearance: Milky turbid suspension, free from impurities (foreign particles)
Microscopic evaluation: Uniform spherical particles,no agglomerates,no fines
Extractable matter (water): ≤ 0.05 %
Cerium: ≤ 10 µg/g
Pressure drop(column: ID=1.6 cm, L=10 cm at 5 ml/min): ≤ 5.0 bar
Particle size (d10): 20 - 28 µm
Particle size (d50): 24 - 34 µm
Particle size (d90): 28 - 38 µm
Colony forming units (TAMC + TYMC): ≤ 100 CFU/ml
Endotoxins: ≤ 1.00 EU/ml
Protein binding capacity (lysozyme): 120 - 180 mg/ml
Functional test (c:d): ≤ 0.15
Functional test (b:a): ≤ 0.15
Functional test: Separation chymotrypsinogen A, cytochrom C and lysozyme

法律信息

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

FRACTOGEL is a registered trademark of Merck KGaA, Darmstadt, Germany

象形图

GHS02

警示用语：

Warning

危险声明

H226

预防措施声明

P210 - P233 - P240 - P241 - P242 - P243

危险分类

Flam. Liq. 3

储存分类代码

3 - Flammable liquids

WGK

WGK 1

闪点(°F)

95.0 °F

闪点(°C)

35 °C

分析证书(COA)

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示例

T1503

货号

25G

包装规格/数量

其它示例：

705578-5MG-PW

PL860-CGA/SHF-1EA

MMYOMAG-74K-13

1000309185

输入内容 1.000309185)

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ABT-702 (4-amino-5-(3-bromophenyl)-7-(6-morpholino-pyridin- 3-yl)pyrido[2,3-d]pyrimidine), a novel orally effective adenosine kinase inhibitor with analgesic and anti-inflammatory properties. II. In vivo characterization in the rat.

E A Kowaluk et al.

The Journal of pharmacology and experimental therapeutics, 295(3), 1165-1174 (2000-11-18)

Adenosine kinase (AK; EC 2.7.1.20) is a key intracellular enzyme regulating intra-and extracellular concentrations of adenosine (ADO), an endogenous neuromodulator, antinociceptive, and anti-inflammatory autocoid. AK inhibition provides a means of potentiating local tissue concentrations of endogenous ADO, and AK inhibitors

The effect of ABT-702, a novel adenosine kinase inhibitor, on the responses of spinal neurones following carrageenan inflammation and peripheral nerve injury.

R Suzuki et al.

British journal of pharmacology, 132(7), 1615-1623 (2001-03-27)

1. Adenosine (ADO) receptor activation modulates sensory transmission in the dorsal horn. Little is known about the circumstances underlying release of the purine. The present study was conducted to investigate the effect of a novel and potent non-nucleoside adenosine kinase

Anti-inflammatory effects of ABT-702, a novel non-nucleoside adenosine kinase inhibitor, in rat adjuvant arthritis.

D L Boyle et al.

The Journal of pharmacology and experimental therapeutics, 296(2), 495-500 (2001-02-13)

Adenosine (ADO) is a homeostatic inhibitory autocoid that is released at sites of inflammation and tissue injury, and exerts anti-inflammatory effects via multiple interactions at ADO receptor subtypes. Inhibition of ADO kinase (AK) increases extracellular ADO concentrations and AK inhibitors

ABT-702 (4-amino-5-(3-bromophenyl)-7-(6-morpholinopyridin-3-yl)pyrido[2, 3-d]pyrimidine), a novel orally effective adenosine kinase inhibitor with analgesic and anti-inflammatory properties: I. In vitro characterization and acute antinociceptive effects in the mouse.

M F Jarvis et al.

The Journal of pharmacology and experimental therapeutics, 295(3), 1156-1164 (2000-11-18)

Adenosine (ADO) is an inhibitory neuromodulator that can increase nociceptive thresholds in response to noxious stimulation. Inhibition of the ADO-metabolizing enzyme adenosine kinase (AK) increases extracellular ADO concentrations at sites of tissue trauma and AK inhibitors may have therapeutic potential

Pharmacological characterization of the chronic constriction injury model of neuropathic pain.

Jean De Vry et al.

European journal of pharmacology, 491(2-3), 137-148 (2004-05-14)

The chronic constriction injury model is a rat model of neuropathic pain based on a unilateral loose ligation of the sciatic nerve. The aim of the present study was to test its sensitivity to various clinically validated and experimental drugs.

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