40085

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In matrix derivatization of trichloroethylene metabolites in human plasma with methyl chloroformate and their determination by solid-phase microextraction-gas chromatography-electron capture detector.

Mohana Krishna Reddy Mudiam et al.

Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 925, 63-69 (2013-03-26)

Trichloroethylene (TCE) is a common industrial chemical that has been widely used as metal degreaser and for many industrial purposes. In humans, TCE is metabolized into dichloroacetic acid (DCA), trichloroacetic acid (TCA) and trichloroethanol (TCOH). A simple and rapid method

Hydrodechlorination of trichloroethylene over Pd supported on swellable organically-modified silica (SOMS)

Sohn H, et al.

Applied Catalysis. B, Environmental, 203, 641-653 (2017)

Assessment of serum S100B and neuron specific enolase levels to evaluate the neurotoxic effects of organic solvent exposure.

Payal Sud et al.

Clinical toxicology (Philadelphia, Pa.), 51(10), 1244-1244 (2013-10-22)

Comparative analysis of the relationship between trichloroethylene metabolism and tissue-specific toxicity among inbred mouse strains: kidney effects.

Hong Sik Yoo et al.

Journal of toxicology and environmental health. Part A, 78(1), 32-49 (2014-11-27)

Trichloroethylene (TCE) is a well-known environmental and occupational toxicant that is classified as carcinogenic to humans based on the epidemiological evidence of an association with higher risk of renal-cell carcinoma. A number of scientific issues critical for assessing human health

Distributions and sea-to-air fluxes of chloroform, trichloroethylene, tetrachloroethylene, chlorodibromomethane and bromoform in the Yellow Sea and the East China Sea during spring.

Zhen He et al.

Environmental pollution (Barking, Essex : 1987), 177, 28-37 (2013-03-08)

Halocarbons including chloroform (CHCl3), trichloroethylene (C2HCl3), tetrachloroethylene (C2Cl4), chlorodibromomethane (CHBr2Cl) and bromoform (CHBr3) were measured in the Yellow Sea (YS) and the East China Sea (ECS) during spring 2011. The influences of chlorophyll a, salinity and nutrients on the distributions

Difficulty of mode of action determination for trichloroethylene: An example of complex interactions of metabolites and other chemical exposures.

Jane C Caldwell et al.

Environmental and molecular mutagenesis, 49(2), 142-154 (2007-11-02)

The mode(s) of action (MOA) of a pollutant for adverse health effects may be dependent on the mixture of metabolites resulting from exposure to a single agent and may also be affected by coexposure to pollutants that have similar targets

Demonstration of compound-specific isotope analysis of hydrogen isotope ratios in chlorinated ethenes.

Tomasz Kuder et al.

Environmental science & technology, 47(3), 1461-1467 (2013-01-09)

High-temperature pyrolysis conversion of organic analytes to H(2) in hydrogen isotope ratio compound-specific isotope analysis (CSIA) is unsuitable for chlorinated compounds such as trichloroethene (TCE) and cis-1,2-dichloroethene (DCE), due to competition from HCl formation. For this reason, the information potential

Trichloroethylene and trichloroethanol-induced formic aciduria and renal injury in male F-344 rats following 12 weeks exposure.

Noreen Yaqoob et al.

Toxicology, 323, 70-77 (2014-06-14)

Trichloroethylene (TCE) is widely used as a cleaning and decreasing agent and has been shown to cause liver tumours in rodents and a small incidence of renal tubule tumours in male rats. The basis for the renal tubule injury is

Hypersensitivity syndrome due to trichloroethylene exposure: a severe generalized skin reaction resembling drug-induced hypersensitivity syndrome.

Hideaki Watanabe

The Journal of dermatology, 38(3), 229-235 (2011-02-24)

The number of patients suffering from trichloroethylene (TCE)-related severe skin disorders with liver dysfunction has been increasing in developing countries in Asia, especially since the mid-1990s. In Japan, five cases of this disease have been reported, but none since the

Identification of the proteins related to SET-mediated hepatic cytotoxicity of trichloroethylene by proteomic analysis.

Xiaohu Ren et al.

Toxicology letters, 227(1), 12-19 (2014-03-19)

Trichloroethylene (TCE) is an effective solvent for a variety of organic materials. Since the wide use of TCE as industrial degreasing of metals, adhesive paint and polyvinyl chloride production, TCE has turned into an environmental and occupational toxicant. Exposure to

Inhibition of Geobacter dechlorinators at elevated trichloroethene concentrations is explained by a reduced activity rather than by an enhanced cell decay.

Jo Philips et al.

Environmental science & technology, 47(3), 1510-1517 (2013-01-04)

Microbial dechlorination of trichloroethene (TCE) is inhibited at elevated TCE concentrations. A batch experiment and modeling analysis were performed to examine whether this self-inhibition is related to an enhanced cell decay or a reduced dechlorination activity at increasing TCE concentrations.

Genetic signature for human risk assessment: lessons from trichloroethylene.

Yih-Horng Shiao

Environmental and molecular mutagenesis, 50(1), 68-77 (2008-11-26)

Trichloroethylene (TCE), an organic solvent commonly used for metal degreasing and as a chemical additive, is a significant environmental contaminant that poses health concerns in humans. The US Environmental Protection Agency (EPA) is currently revising the 2001 TCE human risk

N-Acetylcysteine protects against trichloroethene-mediated autoimmunity by attenuating oxidative stress.

Gangduo Wang et al.

Toxicology and applied pharmacology, 273(1), 189-195 (2013-09-03)

Exposure to trichloroethene (TCE), a ubiquitous environmental contaminant, is known to induce autoimmunity both in humans and animal models. However, mechanisms underlying TCE-mediated autoimmunity remain largely unknown. Previous studies from our laboratory in MRL+/+ mice suggest that oxidative stress may

Uses of and exposure to trichloroethylene in U.S. industry: a systematic literature review.

Berit Bakke et al.

Journal of occupational and environmental hygiene, 4(5), 375-390 (2007-04-25)

This article describes a systematic review of the industrial hygiene literature for uses of trichloroethylene (TCE) in industry for the exposure assessment of two population-based case control studies of brain cancer in the United States. Papers and reports that address

Solvents and Parkinson disease: a systematic review of toxicological and epidemiological evidence.

Edward A Lock et al.

Toxicology and applied pharmacology, 266(3), 345-355 (2012-12-12)

Parkinson disease (PD) is a debilitating neurodegenerative motor disorder, with its motor symptoms largely attributable to loss of dopaminergic neurons in the substantia nigra. The causes of PD remain poorly understood, although environmental toxicants may play etiologic roles. Solvents are

Complement C3a binding to its receptor as a negative modulator of Th2 response in liver injury in trichloroethylene-sensitized mice.

Feng Wang et al.

Toxicology letters, 229(1), 229-239 (2014-07-01)

Trichloroethylene (TCE) is a major occupational health hazard and causes occupational medicamentosa-like dermatitis (OMLDT) and liver damage. Recent evidence suggests immune response as a distinct mode of action for TCE-induced liver damage. This study aimed to explore the role of

Modeling adsorption kinetics of trichloroethylene onto biochars derived from soybean stover and peanut shell wastes.

Mahtab Ahmad et al.

Environmental science and pollution research international, 20(12), 8364-8373 (2013-04-24)

Trichloroethylene (TCE) is one of the most hazardous organic pollutants in groundwater. Biochar produced from agricultural waste materials could serve as a novel carbonaceous adsorbent for removing organic contaminants from aqueous media. Biochars derived from pyrolysis of soybean stover at

Guided cobalamin biosynthesis supports Dehalococcoides mccartyi reductive dechlorination activity.

Jun Yan et al.

Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 368(1616), 20120320-20120320 (2013-03-13)

Dehalococcoides mccartyi strains are corrinoid-auxotrophic Bacteria and axenic cultures that require vitamin B12 (CN-Cbl) to conserve energy via organohalide respiration. Cultures of D. mccartyi strains BAV1, GT and FL2 grown with limiting amounts of 1 µg l(-1) CN-Cbl quickly depleted

Electron donor limitations reduce microbial enhanced trichloroethene DNAPL dissolution: a flux-based analysis using diffusion-cells.

Jo Philips et al.

Chemosphere, 91(1), 7-13 (2012-12-12)

Electron donor limitations likely reduce microbial enhanced trichloroethene (TCE) dense non-aqueous phase liquid (DNAPL) dissolution. This study quantitatively examined the relation between the DNAPL dissolution enhancement and the electron donor supply rate. An experiment used diffusion-cells with a 5.5 cm

Acidification due to microbial dechlorination near a trichloroethene DNAPL is overcome with pH buffer or formate as electron donor: experimental demonstration in diffusion-cells.

Jo Philips et al.

Journal of contaminant hydrology, 147, 25-33 (2013-03-19)

Acidification due to microbial dechlorination of trichloroethene (TCE) can limit the bio-enhanced dissolution of TCE dense non-aqueous phase liquid (DNAPL). This study related the dissolution enhancement of a TCE DNAPL to the pH buffer capacity of the medium and the

Microbiology. Breathing the unbreathable.

Elizabeth Anne Edwards

Science (New York, N.Y.), 346(6208), 424-425 (2014-10-25)

Assessing environmental sustainability of remediation technologies in a life cycle perspective is not so easy.

Mikołaj Owsianiak et al.

Environmental science & technology, 47(3), 1182-1183 (2013-01-18)

Passivation of zero-valent iron by denitrifying bacteria and the impact on trichloroethene reduction in groundwater.

Liang Chen et al.

Water science and technology : a journal of the International Association on Water Pollution Research, 67(6), 1254-1259 (2013-03-20)

Zero-valent iron (ZVI) application in groundwater remediation is limited by its vulnerability to passivation, which significantly decreases its surface reactivity. Both biological and chemical processes can potentially passivate ZVI, although the understanding of biological passivation is limited. This study was

Pleiotropic effects of the trichloroethylene-associated P81S VHL mutation on metabolism, apoptosis, and ATM-mediated DNA damage response.

Michelle C Desimone et al.

Journal of the National Cancer Institute, 105(18), 1355-1364 (2013-08-31)

The risk relevance of the P81S von Hippel-Lindau (VHL) gene hotspot mutation identified in clear cell renal cell carcinoma from individuals exposed occupationally to trichloroethylene (TCE) is not known. VHL mutations in hereditary VHL syndrome strongly correlate with phenotypic associations

Trichloroethylene and cancer.

Mark P Purdue

Journal of the National Cancer Institute, 105(12), 844-846 (2013-06-01)

Inhibition of microbial trichloroethylene dechlorination [corrected] by Fe (III) reduction depends on Fe mineralogy: a batch study using the bioaugmentation culture KB-1.

Laiby Paul et al.

Water research, 47(7), 2543-2554 (2013-03-16)

Microbial reductive dechlorination of trichloroethylene (TCE) in groundwater can be stimulated by adding of electron donors. However, side reactions such as Fe (III) reduction competes with this reaction. This study was set-up to relate the inhibition of microbial TCE dechlorination

TCE, designated a known carcinogen, now the focus of ongoing research.

Caroline McNeil

Journal of the National Cancer Institute, 105(20), 1518-1519 (2013-10-02)

Trichloroethylene and ocular malformations: analysis of extant literature.

Amy Lavin Williams et al.

International journal of toxicology, 27(1), 81-95 (2008-02-23)

An evaluation of the scientific literature for trichloroethylene (TCE) identified two reports of ocular defects, specifically microphthalmia/anophthalmia, following prenatal TCE exposure in rats. Herein, these reports are analyzed in detail and interpreted within the context of other developmental TCE exposure

Micronucleus induction by oxidative metabolites of trichloroethylene in cultured human peripheral blood lymphocytes: a comparative genotoxicity study.

Meenu Varshney et al.

Environmental science and pollution research international, 20(12), 8709-8716 (2013-05-31)

The genotoxic effects of oxidative metabolites of trichloroethylene (TCE), namely chloral hydrate, trichloroacetic acid (TCA), dichloroacetic acid (DCA), and trichloroethanol (TCEOH) were examined in human peripheral blood lymphocytes. In this context, lymphocytes were exposed in vitro to 25, 50, and

New insights into von Hippel-Lindau function highlighted by investigation of the trichloroethylene-induced p.P81S hotspot mutation.

Len Neckers et al.

Journal of the National Cancer Institute, 105(18), 1339-1340 (2013-08-31)

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