F3929

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Effects of FSHR polymorphisms on premature ovarian insufficiency in human beings: a meta-analysis.

Wenling Huang et al.

Reproductive biology and endocrinology : RB&E, 17(1), 80-80 (2019-10-21)

Whether follicle-stimulating hormone receptor (FSHR) polymorphisms are implicated in premature ovarian insufficiency (POI) remains controversial. Thus, we performed this study to explore correlation between FSHR polymorphisms and POI in human beings. Literature retrieve was conducted in PubMed, Medline, Embase and

Current concepts of follicle-stimulating hormone receptor gene regulation.

Jitu W George et al.

Biology of reproduction, 84(1), 7-17 (2010-08-27)

Follicle-stimulating hormone (FSH), a pituitary glycoprotein hormone, is an integral component of the endocrine axis that regulates gonadal function and fertility. To transmit its signal, FSH must bind to its receptor (FSHR) located on Sertoli cells of the testis and

The follicle-stimulating hormone receptor: biochemistry, molecular biology, physiology, and pathophysiology.

M Simoni et al.

Endocrine reviews, 18(6), 739-773 (1997-12-31)

Follicle-stimulating hormone receptors expression in ovine corpora lutea during luteal phase: effect of nutritional plane and follicle-stimulating hormone treatment.

A T Grazul-Bilska et al.

Domestic animal endocrinology, 71, 106391-106391 (2019-11-16)

Corpus luteum (CL), a transient endocrine gland critical for reproductive cyclicity and pregnancy maintenance, is controlled by numerous regulatory factors. Although LH is widely recognized as the major regulator, other factors may also affect luteal functions. It has been demonstrated

Association study between FSHR Ala307Thr and Ser680Asn variants and polycystic ovary syndrome (PCOS) in Northern Chinese Han women.

Lingyuan Fu et al.

Journal of assisted reproduction and genetics, 30(5), 717-721 (2013-03-29)

Polycystic ovary syndrome (PCOS) is a common complex genetic endocrinopathy. It has high heritability, and twin studies indicate that it is a complex polygenic disorder. Searching for major genes of PCOS is crucial to clarify its molecular pathogenesis. A previous

Bone marrow-derived mesenchymal stem cells combined with gonadotropin therapy restore postnatal oogenesis of chemo-ablated ovaries in rats via enhancing very small embryonic-like stem cells.

Nesrine Ebrahim et al.

Stem cell research & therapy, 12(1), 517-517 (2021-09-29)

Very small embryonic-like stem cells (VSELs) are a rare population within the ovarian epithelial surface. They contribute to postnatal oogenesis as they have the ability to generate immature oocytes and resist the chemotherapy. These cells express markers of pluripotent embryonic

The expression of the follicle-stimulating hormone receptor in spermatogenesis.

Leslie L Heckert et al.

Recent progress in hormone research, 57, 129-148 (2002-05-23)

Results from experiments using mouse models suggest that the role of follicle-stimulating hormone (FSH) in spermatogenesis is the regulation of Sertoli cell proliferation and, ultimately, the size and spermatogenic capacity of the testis. The regulation of the expression of the

Postnatal exposure to chromium through mother's milk accelerates follicular atresia in F1 offspring through increased oxidative stress and depletion of antioxidant enzymes.

Jone A Stanley et al.

Free radical biology & medicine, 61, 179-196 (2013-03-09)

Hexavalent chromium, CrVI, is a heavy metal endocrine disruptor, known as a mutagen, teratogen, and a group A carcinogen. Environmental contamination with CrVI, including drinking water, has been increasing in more than 30 cities in the United States. CrVI is

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