HPA001853

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关键词:'HPA001853'

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The role of Sp1 and Sp3 in normal and cancer cell biology.

Lin Li et al.

Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft, 192(5), 275-283 (2010-09-03)

Sp1 and Sp3 are transcription factors expressed in all mammalian cells. These factors are involved in regulating the transcriptional activity of genes implicated in most cellular processes. Dysregulation of Sp1 and Sp3 is observed in many cancers and diseases. Due

Chromosome 12q13. 13q13. 13 microduplication and microdeletion: a case report and literature review

Hu J, et al.

Molecular Cytogenetics, 10(1), 24-24 (2017)

Identification of a novel Sp1 splice variant as a strong transcriptional activator.

Vittoria Infantino et al.

Biochemical and biophysical research communications, 412(1), 86-91 (2011-07-30)

The transcription factor Sp1 regulates expression of numerous genes involved in many cellular processes. Different post-transcriptional modifications can influence the transcriptional control activity and stability of Sp1. In addition to these modifications, alternative splicing isoforms may also be the basis

Inhibition of the invasion and metastasis of mammary carcinoma cells by NBD peptide targeting S100A4 via the suppression of the Sp1/MMP-14 axis

Takenaga K, et al.

International Journal of Oncology, 58(3), 397-408 null

Functional interactions between Sp1 or Sp3 and the helicase-like transcription factor mediate basal expression from the human plasminogen activator inhibitor-1 gene.

H Ding et al.

The Journal of biological chemistry, 274(28), 19573-19580 (1999-07-03)

Basal expression of the human plasminogen activator inhibitor-1 (PAI-1) is mediated by a promoter element named B box that binds the helicase-like transcription factor (HLTF), homologous to SNF/SWI proteins. Electrophoretic mobility shift assays performed on a set of B box

Inhibition of the invasion and metastasis of mammary carcinoma cells by NBD peptide targeting S100A4 via the suppression of the Sp1/MMP‑14 axis.

Keizo Takenaga et al.

International journal of oncology, 58(3), 397-408 (2021-03-03)

A synthetic peptide that blocks the interaction between the metastasis‑enhancing calcium‑binding protein, S100A4, and its effector protein, methionine aminopeptidase 2 (MetAP2) (the NBD peptide), was previously demonstrated to inhibit the angiogenesis of endothelial cells, leading to the regression of human prostate

Celecoxib inhibits vascular endothelial growth factor expression in and reduces angiogenesis and metastasis of human pancreatic cancer via suppression of Sp1 transcription factor activity.

Daoyan Wei et al.

Cancer research, 64(6), 2030-2038 (2004-03-18)

The aggressive biology of human pancreatic adenocarcinoma has been linked with overexpression of vascular endothelial growth factor (VEGF). Constitutive activation of the transcription factor Sp1 plays a critical role in VEGF overexpression. Recent studies indicated that celecoxib, a selective cyclooxygenase-2

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