SBR00013

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Actinomycin D induces apoptosis and inhibits growth of pancreatic cancer cells.

J Kleeff et al.

International journal of cancer, 86(3), 399-407 (2000-04-13)

Pancreatic cancer cells are usually resistant to apoptosis induced by cytotoxic drugs, by activation of surface receptors such as Fas and TNF receptor or by serum or growth factor withdrawal. Actinomycin D (actD) is an inhibitor of RNA synthesis and

Hsa_circ_001659 serves as a novel diagnostic and prognostic biomarker for colorectal cancer.

Baoyu He et al.

Biochemical and biophysical research communications, 551, 100-106 (2021-03-17)

Colorectal cancer (CRC) is prevalent worldwide and novel diagnostic and prognostic biomarkers are needed to improve precision medicine. Circular RNAs (circRNAs) are currently being considered as emerging tumor biomarkers. Herein, we aimed to explore the possible clinical application of circRNAs

Long non-coding RNA CASC2 enhances irradiation-induced endoplasmic reticulum stress in NSCLC cells through PERK signaling.

Zhouli Ding et al.

3 Biotech, 10(10), 449-449 (2020-10-17)

Radiotherapy is instrumental in the treatment of inoperable non-small cell lung cancer (NSCLC). Studies have revealed that radiotherapy induces endoplasmic reticulum (ER) stress, which consequently induces apoptosis and sensitization of cancer cells. A recent study has revealed that long non-coding

Goodman and Gilman?s The Pharmacological Basis of Therapeutics, 1240-1241 (1990)

Reynolds, J.E.F, Ed.

Martindale: The Extra Pharmacopoeia, 473-473 (1993)

Interaction of actinomycin D with yeast ribosomal RNA.

L W Waltschewa

FEBS letters, 111(1), 179-180 (1980-02-25)

Sensitivity of normal and mutant strains of Escherichia coli to actinomycin-D.

A P Singh et al.

Canadian journal of microbiology, 18(6), 909-915 (1972-06-01)

Actinomycin D inhibition of DNA strand transfer reactions catalyzed by HIV-1 reverse transcriptase and nucleocapsid protein.

W R Davis et al.

Biochemistry, 37(40), 14213-14221 (1998-10-07)

Actinomycin D was found to be a potent inhibitor of HIV-1 reverse transcriptase catalyzed DNA strand transfer reactions. Using an oligonucleotide model system, actinomycin D inhibition of DNA strand transfer was examined to elucidate the mechanism of inhibition and further

Actinomycin D inhibits human immunodeficiency virus type 1 minus-strand transfer in in vitro and endogenous reverse transcriptase assays.

J Guo et al.

Journal of virology, 72(8), 6716-6724 (1998-07-11)

In this report we demonstrate that human immunodeficiency virus type 1 (HIV-1) minus-strand transfer, assayed in vitro and in endogenous reactions, is greatly inhibited by actinomycin D. Previously we showed that HIV-1 nucleocapsid (NC) protein (a nucleic acid chaperone catalyzing

Inhibiting eukaryotic transcription: Which compound to choose? How to evaluate its activity?

Olivier Bensaude

Transcription, 2(3), 103-108 (2011-09-17)

This review first discusses ways in which we can evaluate transcription inhibition, describe changes in nuclear structure due to transcription inhibition, and report on genes that are paradoxically stimulated by transcription inhibition. Next, it summarizes the characteristics and mechanisms of

The binding of actinomycin D and F to bacterial DNA.

L F Cavalieri et al.

Biochimica et biophysica acta, 166(3), 722-725 (1968-10-29)

Nerve growth factor and epidermal growth factor rescue PC12 cells from programmed cell death induced by etoposide: distinct modes of protection against cell death by growth factors and a protein-synthesis inhibitor.

M Nakajima et al.

Neuroscience letters, 176(2), 161-164 (1994-08-01)

A rat pheochromocytoma cell line (PC12 cells) died within 24 h in the presence of etoposide (1-40 micrograms/ml), an inhibitor of topoisomerase II. This cytotoxic effect was prevented by either nerve growth (NGF) or epidermal growth factor (EGF). Cycloheximide and

Dawson, R.M.C., et al., Eds.

Data for Biochemical Research, 256-257 (1986)

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