质量水平
方案
98%
表单
liquid
折射率
n20/D 1.552 (lit.)
沸点
260-270 °C (lit.)
密度
1.055 g/mL at 25 °C (lit.)
储存温度
2-8°C
SMILES字符串
CCOC(=O)C#Cc1ccccc1
InChI
1S/C11H10O2/c1-2-13-11(12)9-8-10-6-4-3-5-7-10/h3-7H,2H2,1H3
InChI key
ACJOYTKWHPEIHW-UHFFFAOYSA-N
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储存分类代码
10 - Combustible liquids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves
G S Cameron et al.
The Journal of investigative dermatology, 99(2), 189-192 (1992-08-01)
The dorsal skin of hairless mice (Skh:HR-1) was treated with multiple applications of acetone, 12-O-tetradecanoyl-phorbol-13-acetate (TPA) or ethyl phenylpropionate (EPP) two times per week, or exposed to ultraviolet radiation (UVR) three times per week for treatment periods up to 16
B H Shearer et al.
Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology, 23(4), 149-155 (1994-04-01)
The effects of four different hyperplastic agents and of the carcinogen DMBA on cytokeratin expression in hamster cheek pouch epithelia were compared. Reversible hyperplasia was produced by the application of either oil of turpentine, vitamin A or TPA. No hyperplastic
S K Gilmour et al.
Carcinogenesis, 13(1), 51-56 (1992-01-01)
Single applications of 12-O-tetradecanoylphorbol-13-acetate (TPA), mezerein or ethyl phenylpropriolate (EPP) to mouse skin at appropriate doses cause similar degrees of hyperplasia and comparable levels of induction of epidermal ornithine decarboxylase (ODC) activity. Multiple (n = 5) treatments with these agents
E Patrick et al.
Toxicology and applied pharmacology, 81(3 Pt 1), 476-490 (1985-12-01)
The possibility that chemicals induce skin irritation by multiple mechanisms was studied in laboratory mice. The time course and dose response to topical application of phenol, croton oil, benzalkonium chloride, ethyl phenylpropiolate (EPP), and methyl salicylate were compared. The responses
C S Baxter et al.
Carcinogenesis, 10(10), 1855-1861 (1989-10-01)
Responses of various cells of the epidermis and dermis to topically applied agents have been implicated in the mechanism of multistage mouse tumorigenesis. These responses have been discussed almost entirely in the context of a single promoter treatment, although tumor
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